PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32701561-0	2020	Icaritin inhibits lung cancer-induced osteoclastogenesis by suppressing the expression of IL-6 and TNF-a and through AMPK/mTOR signaling pathway.	icaritin	0-8	protein kinase AMP-activated catalytic subunit alpha 2	Homo sapiens	117-121	
32701561-9	2020	Furthermore, ICT decreased the levels of IL-6 and TNF-alpha in osteoclasts, while the AMPK inhibitor compound C significantly abolished the inhibitory effects of ICT on IL-6 and TNF-alpha.	icaritin	162-165	protein kinase AMP-activated catalytic subunit alpha 2	Homo sapiens	86-90	
34221923-0	2021	Icaritin Attenuates Lipid Accumulation by Increasing Energy Expenditure and Autophagy Regulated by Phosphorylating AMPK.	icaritin	0-8	protein kinase AMP-activated catalytic subunit alpha 2	Homo sapiens	115-119	
34221923-9	2021	Icaritin enhanced glucose uptake, decreased adenosine triphosphate content, and activated the AMPK signaling pathway in 3T3-L1 adipocytes and C2C12 myotubes.	icaritin	0-8	protein kinase AMP-activated catalytic subunit alpha 2	Homo sapiens	94-98	
34221923-12	2021	Conclusions: Icaritin attenuates lipid accumulation by increasing energy expenditure and regulating autophagy by activating the AMPK pathway.	icaritin	13-21	protein kinase AMP-activated catalytic subunit alpha 2	Homo sapiens	128-132