PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
10543765-1	1999	In vitro treatment with clarithromycin inhibited the expression of the matrix metalloproteinase-9, transforming growth factor beta, and tumor necrosis factor alpha genes in 13762NF rat mammary adenocarcinoma cells.	Clarithromycin	24-38	matrix metallopeptidase 9	Rattus norvegicus	71-97	
28070612-8	2017	In vivo, chemically-induced HCC rat model proved that clarithromycin suppressed HCC via induction of apoptosis through up-regulation of both extrinsic/intrinsic apoptotic pathways" proteins (TNFR1, cleaved caspase-3, and Bax with an increased Bax/Bcl-2 ratio) along with MMP-9 normalization.	Clarithromycin	54-68	matrix metallopeptidase 9	Rattus norvegicus	271-276	
26156575-1	2015	The aim of this study was to investigate the effect of clarithromycin in rat endometriosis and its association with matrix metalloproteinase-9 (MMP-9) expression.	Clarithromycin	55-69	matrix metallopeptidase 9	Rattus norvegicus	116-142	
26156575-1	2015	The aim of this study was to investigate the effect of clarithromycin in rat endometriosis and its association with matrix metalloproteinase-9 (MMP-9) expression.	Clarithromycin	55-69	matrix metallopeptidase 9	Rattus norvegicus	144-149	
20350988-3	2010	However, little is known about the effect of CAM in myocarditis via MMPs.	Clarithromycin	45-48	matrix metallopeptidase 9	Rattus norvegicus	68-72	
20350988-10	2010	CONCLUSIONS: Early CAM treatment is effective to attenuate myocarditis by suppressing MMP-9.	Clarithromycin	19-22	matrix metallopeptidase 9	Rattus norvegicus	86-91