PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 10543765-1 1999 In vitro treatment with clarithromycin inhibited the expression of the matrix metalloproteinase-9, transforming growth factor beta, and tumor necrosis factor alpha genes in 13762NF rat mammary adenocarcinoma cells. Clarithromycin 24-38 matrix metallopeptidase 9 Rattus norvegicus 71-97 28070612-8 2017 In vivo, chemically-induced HCC rat model proved that clarithromycin suppressed HCC via induction of apoptosis through up-regulation of both extrinsic/intrinsic apoptotic pathways" proteins (TNFR1, cleaved caspase-3, and Bax with an increased Bax/Bcl-2 ratio) along with MMP-9 normalization. Clarithromycin 54-68 matrix metallopeptidase 9 Rattus norvegicus 271-276 26156575-1 2015 The aim of this study was to investigate the effect of clarithromycin in rat endometriosis and its association with matrix metalloproteinase-9 (MMP-9) expression. Clarithromycin 55-69 matrix metallopeptidase 9 Rattus norvegicus 116-142 26156575-1 2015 The aim of this study was to investigate the effect of clarithromycin in rat endometriosis and its association with matrix metalloproteinase-9 (MMP-9) expression. Clarithromycin 55-69 matrix metallopeptidase 9 Rattus norvegicus 144-149 20350988-3 2010 However, little is known about the effect of CAM in myocarditis via MMPs. Clarithromycin 45-48 matrix metallopeptidase 9 Rattus norvegicus 68-72 20350988-10 2010 CONCLUSIONS: Early CAM treatment is effective to attenuate myocarditis by suppressing MMP-9. Clarithromycin 19-22 matrix metallopeptidase 9 Rattus norvegicus 86-91