PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
27296639-2	2016	In this study, application of the group I mGlu receptor agonist, DHPG, caused LTD of NMDA-EPSCs (DHPG-LTDNMDA) of the Schaffer collateral, but not of NMDA-EPSCs of the temperoammonic pathway onto CA1 neurons of the hippocampus.	3,4-dihydroxyphenylglycol	65-69	carbonic anhydrase 1	Homo sapiens	196-199	
10530820-0	1999	An investigation into signal transduction mechanisms involved in DHPG-induced LTD in the CA1 region of the hippocampus.	3,4-dihydroxyphenylglycol	65-69	carbonic anhydrase 1	Homo sapiens	89-92	
16023152-5	2005	Results show that short (10 min) DHPG treatment did not result in neuroprotection but significantly depressed field synaptic potentials (fEPSP) in the Schaffer collateral-CA1 pathway.	3,4-dihydroxyphenylglycol	33-37	carbonic anhydrase 1	Homo sapiens	171-174	
16023152-8	2005	Voltage-clamp measurements of spontaneous miniature excitatory post-synaptic currents (EPSCs) recorded in CA1 neurons from cultures treated with DHPG (10 microM, 2 h) showed a significant reduction of the EPSC amplitude in DHPG-treated but not control (untreated) cultures.	3,4-dihydroxyphenylglycol	145-149	carbonic anhydrase 1	Homo sapiens	106-109	
16023152-8	2005	Voltage-clamp measurements of spontaneous miniature excitatory post-synaptic currents (EPSCs) recorded in CA1 neurons from cultures treated with DHPG (10 microM, 2 h) showed a significant reduction of the EPSC amplitude in DHPG-treated but not control (untreated) cultures.	3,4-dihydroxyphenylglycol	223-227	carbonic anhydrase 1	Homo sapiens	106-109