PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33288764-4	2020	Here, we show that gemcitabine, a standard chemotherapy for various solid tumors, triggers hallmark immunostimualtory DAMP release (e.g., calreticulin, HSP70, and HMGB1); however, is unable to induce ICD.	gemcitabine	19-30	calreticulin	Homo sapiens	138-150	
33591948-6	2021	Knockdown of CRT inhibited cell proliferation, induced apoptosis, arrested cell cycle and resulted in decreased resistance to gemcitabine, which was mediated by the inactivation of the PI3K/Akt pathway.	gemcitabine	126-137	calreticulin	Homo sapiens	13-16	
24264800-8	2014	In addition, CRT knockdown significantly decreased pho-ERK expression and cell chemoresistance independent of activated p53 and caspase-3-related apoptosis in gemcitabine- or oxaliplatin-treated Capan-2 cells.	gemcitabine	159-170	calreticulin	Homo sapiens	13-16	
32161598-4	2020	In our study, we observed that low-dose gemcitabine treatment enhanced the immunogenicity of lung cancer by increasing the exposure of calreticulin, high mobility group box 1, and upregulating expression of NKG2D ligands.	gemcitabine	40-51	calreticulin	Homo sapiens	135-147