PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 33288764-4 2020 Here, we show that gemcitabine, a standard chemotherapy for various solid tumors, triggers hallmark immunostimualtory DAMP release (e.g., calreticulin, HSP70, and HMGB1); however, is unable to induce ICD. gemcitabine 19-30 calreticulin Homo sapiens 138-150 32161598-4 2020 In our study, we observed that low-dose gemcitabine treatment enhanced the immunogenicity of lung cancer by increasing the exposure of calreticulin, high mobility group box 1, and upregulating expression of NKG2D ligands. gemcitabine 40-51 calreticulin Homo sapiens 135-147 24264800-8 2014 In addition, CRT knockdown significantly decreased pho-ERK expression and cell chemoresistance independent of activated p53 and caspase-3-related apoptosis in gemcitabine- or oxaliplatin-treated Capan-2 cells. gemcitabine 159-170 calreticulin Homo sapiens 13-16 33591948-6 2021 Knockdown of CRT inhibited cell proliferation, induced apoptosis, arrested cell cycle and resulted in decreased resistance to gemcitabine, which was mediated by the inactivation of the PI3K/Akt pathway. gemcitabine 126-137 calreticulin Homo sapiens 13-16