PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23420263-9	2013	BID and FOS, which are known apoptosis activators, were upregulated by lenalidomide treatment, whereas NKX2-1, which is used as an immunohistochemistry marker for NSCLC, was downregulated.	Lenalidomide	71-83	BH3 interacting domain death agonist	Homo sapiens	0-3	
19965642-7	2010	In CLL patients receiving lenalidomide, similar evidence of CD154 activation is observed including BID, DR5, and p73 induction and also development of anti-ROR1 tumor-directed antibodies.	Lenalidomide	26-38	BH3 interacting domain death agonist	Homo sapiens	99-102