PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23884819-13	2013	The combinations of verapamil with either carbenoxolone or pioglitazone caused further reduction in ulcer index, gastric acid output and 15-PGDH activity (p < 0.05), while causing further increase in gastric barrier mucus (p < 0.05) compared to their respective individual treatment group.	Pioglitazone	59-71	15-hydroxyprostaglandin dehydrogenase	Rattus norvegicus	137-144	
23884819-0	2013	15-PGDH inhibitors: the antiulcer effects of carbenoxolone, pioglitazone and verapamil in indomethacin induced peptic ulcer rats.	Pioglitazone	60-72	15-hydroxyprostaglandin dehydrogenase	Rattus norvegicus	0-7	
23884819-3	2013	The aim of the present study is to investigate the inhibitory effect of carbenoxolone, pioglitazone and verapamil on 15-PGDH enzyme.	Pioglitazone	87-99	15-hydroxyprostaglandin dehydrogenase	Rattus norvegicus	117-124	
23884819-11	2013	The rats pretreated with carbenoxolone, pioglitazone, verapamil had reduced ulcer index, gastric acid output and 15-PGDH activity (p < 0.05) compared to either indomethacin group or the negative control group.	Pioglitazone	40-52	15-hydroxyprostaglandin dehydrogenase	Rattus norvegicus	113-120	
23884819-14	2013	CONCLUSIONS: The antiulcer properties of pioglitazone and verapamil are, in part, consequences of their inhibitory effect on the enzyme 15-PGDH, responsible for PGs degradation, and the resultant prolongation of PGE2 biological activity in rat stomach mucosa.	Pioglitazone	41-53	15-hydroxyprostaglandin dehydrogenase	Rattus norvegicus	136-143