PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26012717-2	2015	Using NPCs cultured from PKCepsilon or beta-arrestin2 knockout mice and the MAPK/ERK kinase inhibitor U0126, we demonstrate that regulation of NPC differentiation via the miR-181a/Prox1/Notch1 pathway exhibits ligand-dependent selectivity.	U 0126	102-107	microRNA 181a-2	Mus musculus	171-179	
30938884-10	2019	When we knocked down miR-181a and then treated cells with U0126 before ox-LDL stimulation, we found that U0126 reversed the increased activation of the MEK/ERK/NF-kappaB pathway and upregulation of NLRP3 inflammasome-related proteins (NLRP3, caspase-1, IL-18, IL-1beta) that resulted from miR-181a knockdown.	U 0126	58-63	microRNA 181a-2	Mus musculus	289-297	
30938884-10	2019	When we knocked down miR-181a and then treated cells with U0126 before ox-LDL stimulation, we found that U0126 reversed the increased activation of the MEK/ERK/NF-kappaB pathway and upregulation of NLRP3 inflammasome-related proteins (NLRP3, caspase-1, IL-18, IL-1beta) that resulted from miR-181a knockdown.	U 0126	105-110	microRNA 181a-2	Mus musculus	21-29	
30938884-10	2019	When we knocked down miR-181a and then treated cells with U0126 before ox-LDL stimulation, we found that U0126 reversed the increased activation of the MEK/ERK/NF-kappaB pathway and upregulation of NLRP3 inflammasome-related proteins (NLRP3, caspase-1, IL-18, IL-1beta) that resulted from miR-181a knockdown.	U 0126	105-110	microRNA 181a-2	Mus musculus	289-297