PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25279705-10	2014	Taken together, the HDAC1/miR-200b/Suz-12-E-cadherin signaling might account for maintenance of CSCs and formation of chemoresistant phenotype in docetaxel-resistant LAD cells.	Docetaxel	146-155	histone deacetylase 1	Homo sapiens	20-25	
24830600-6	2014	Furthermore, silencing of HDAC1/4 suppressed cell proliferation, promoted cell apoptosis, induced G2/M cell cycle arrest and ultimately reversed in vitro and in vivo chemoresistance of docetaxel-resistant LAD cells, at least partially in a miR-200b-dependent manner.	Docetaxel	185-194	histone deacetylase 1	Homo sapiens	26-31	
24830600-8	2014	HDAC1/4 levels in docetaxel-insensitive human LAD tissues, inversely correlated with miR-200b, were upregulated compared with docetaxel-sensitive tissues.	Docetaxel	18-27	histone deacetylase 1	Homo sapiens	0-5	
24830600-8	2014	HDAC1/4 levels in docetaxel-insensitive human LAD tissues, inversely correlated with miR-200b, were upregulated compared with docetaxel-sensitive tissues.	Docetaxel	126-135	histone deacetylase 1	Homo sapiens	0-5	
24830600-9	2014	Taken together, our findings suggest that the HDAC1/4/Sp1/miR-200b/E2F3 pathway is responsible for chemoresistance of docetaxel-resistant LAD cells.	Docetaxel	118-127	histone deacetylase 1	Homo sapiens	46-51