PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
16821082-0	2007	Possible involvement of CCT5, RGS3, and YKT6 genes up-regulated in p53-mutated tumors in resistance to docetaxel in human breast cancers.	Docetaxel	103-112	YKT6 v-SNARE homolog	Homo sapiens	40-44	
16821082-8	2007	Of these 13 genes, mRNA expression of CCT5, RGS3, and YKT6 was significantly up-regulated in p53-mutated tumors and associated with a low response rate to docetaxel.	Docetaxel	155-164	YKT6 v-SNARE homolog	Homo sapiens	54-58	
16821082-9	2007	Treatment of MCF-7 cells with siRNA specific for CCT5, RGS3, or YKT6 resulted in a significant enhancement of docetaxel-induced apoptosis.	Docetaxel	110-119	YKT6 v-SNARE homolog	Homo sapiens	64-68	
16821082-10	2007	CONCLUSIONS: CCT5, RGS3, and YKT6 mRNA expressions, which are up-regulated in p53-mutated breast tumors, might be implicated in resistance to docetaxel and clinically useful in identifying the subset of breast cancer patients who may or may not benefit from docetaxel treatment.	Docetaxel	142-151	YKT6 v-SNARE homolog	Homo sapiens	29-33	
16821082-10	2007	CONCLUSIONS: CCT5, RGS3, and YKT6 mRNA expressions, which are up-regulated in p53-mutated breast tumors, might be implicated in resistance to docetaxel and clinically useful in identifying the subset of breast cancer patients who may or may not benefit from docetaxel treatment.	Docetaxel	258-267	YKT6 v-SNARE homolog	Homo sapiens	29-33