PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
27219337-4	2016	UA treatment (6-9 h) strongly blocked the survival AKT/GSK3beta/beta-catenin signalling pathway, which led to a concomitant reduction of the anti-apoptotic proteins c-Myc and p21, altogether resulting in the activation of intrinsic apoptosis.	ursolic acid	0-2	H3 histone pseudogene 16	Homo sapiens	175-178	
27219337-5	2016	Interestingly, UA at low concentrations (10-15 muM) enhanced the antitumoral effects of DXR by up to 2-fold, while in parallel inhibiting DXR-induced AKT activation and p21 expression, two proteins implicated in antitumoral drug resistance and cell survival.	ursolic acid	15-17	H3 histone pseudogene 16	Homo sapiens	169-172	
34141150-6	2021	Ursolic acid treatment downregulated the gene expression of survival factors BCL-2, SURVIVIN, NFKB and SP1, while upregulated the growth-restricting genes BAX, P21 and P53.	ursolic acid	0-12	H3 histone pseudogene 16	Homo sapiens	160-163