PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 14718582-1 2004 This study aimed to determine the mechanism(s) by which 1,4-dihydropyridine Ca2+ channel blockers (DHPs) enhance the binding of neurotensin (NT) to prostate cancer PC3 cells and inhibit NT-induced inositol phosphate formation. Inositol Phosphates 197-215 deoxyhypusine synthase Homo sapiens 99-103 14718582-4 2004 By varying [Ca2+] and testing the effects of stimulators and inhibitors of Ca2+ influx and internal Ca2+ release, we determined that although DHPs may have inhibited inositol phosphate formation partly by blocking Ca2+ influx, the effect on NT binding was Ca2+-independent. Inositol Phosphates 166-184 deoxyhypusine synthase Homo sapiens 142-146