PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23620203-5	2013	Stress-induced O-GlcNAcylation appears to promote cell/tissue survival by regulating a multitude of biological processes including: the phosphoinositide 3-kinase/Akt pathway, heat shock protein expression, calcium homeostasis, levels of reactive oxygen species, ER stress, protein stability, mitochondrial dynamics, and inflammation.	o-glcnacylation	15-30	AKT serine/threonine kinase 1	Homo sapiens	162-165	
35296731-0	2022	Increased O-GlcNAcylation promotes IGF-1 receptor/PhosphatidyI Inositol-3 kinase/Akt pathway in cervical cancer cells.	o-glcnacylation	10-25	AKT serine/threonine kinase 1	Homo sapiens	81-84	
34941261-8	2022	We find that O-GlcNAcylation at Ser473 and phosphorylation at Tyr474 can also partially increase Akt"s kinase activity toward both peptide and protein substrates.	o-glcnacylation	13-28	AKT serine/threonine kinase 1	Homo sapiens	97-100	
33651993-10	2021	It is suggested that the acute cold exposure of piglets induced a sequential change in the level of O-GlcNAcylation, which may be one of the factors mediating liver cell apoptosis and glucose metabolism regulation by the O-GlcNAc/AKT pathway.	o-glcnacylation	100-115	AKT serine/threonine kinase 1	Homo sapiens	230-233	
32892442-9	2020	Moreover, OSMI-1 further promoted HBV replication by inducing autophagosome formation via inhibiting the O-GlcNAcylation of Akt and mTOR.	o-glcnacylation	105-120	AKT serine/threonine kinase 1	Homo sapiens	124-127	
32538734-0	2020	Elevated O-GlcNAcylation Promotes Malignant Phenotypes of Hypopharyngeal Squamous Cell Carcinoma by Stabilizing Nrf2 through Regulation of the PI3K/Akt Pathway.	o-glcnacylation	9-24	AKT serine/threonine kinase 1	Homo sapiens	148-151