PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
16102732-6	2005	In addition, RA reversed the downregulations of GSH, SOD and Bcl-2 by ADR.	rosmarinic acid	13-15	BCL2, apoptosis regulator	Rattus norvegicus	61-66	
29376419-2	2018	Pretreatment with RA significantly suppressed the generation of ROS, protected the morphological changes of cells, decrease the ratio of cell apoptosis, down-regulated the level of caspase-3, caspase-9, Bax/Bcl-2, and up-regulated the level of p-PI3K.	rosmarinic acid	18-20	BCL2, apoptosis regulator	Rattus norvegicus	207-212	
24085626-4	2014	Additionally, pretreatment with RA potentiated the decrease of Bcl-2 expression and attenuated the increase of Caspase-3 expression by CoCl2.	rosmarinic acid	32-34	BCL2, apoptosis regulator	Rattus norvegicus	63-68	
24085626-6	2014	CoCl2-induced increase of Bax/Bcl-2 ratio and Caspase-3 expression was attenuated by RA, NAC and SB203580 (p38MAPK inhibitor).	rosmarinic acid	85-87	BCL2, apoptosis regulator	Rattus norvegicus	30-35	
22205146-9	2012	Moreover, RA suppressed the increased ratio of Bax/Bcl-2 at gene level induced by 6-OHDA.	rosmarinic acid	10-12	BCL2, apoptosis regulator	Rattus norvegicus	51-56	
22205146-10	2012	This indicates that the neurorescue effects of RA against 6-ODHA-induced degeneration of the nigrostriatal dopaminergic system were achieved by decreasing nigral iron levels and regulating the ratio of Bcl-2/Bax gene expression.	rosmarinic acid	47-49	BCL2, apoptosis regulator	Rattus norvegicus	202-207