PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
29376419-2	2018	Pretreatment with RA significantly suppressed the generation of ROS, protected the morphological changes of cells, decrease the ratio of cell apoptosis, down-regulated the level of caspase-3, caspase-9, Bax/Bcl-2, and up-regulated the level of p-PI3K.	rosmarinic acid	18-20	BCL2 associated X, apoptosis regulator	Rattus norvegicus	203-206	
24085626-6	2014	CoCl2-induced increase of Bax/Bcl-2 ratio and Caspase-3 expression was attenuated by RA, NAC and SB203580 (p38MAPK inhibitor).	rosmarinic acid	85-87	BCL2 associated X, apoptosis regulator	Rattus norvegicus	26-29	
27884046-6	2017	Administration of rosmarinic acid and lycopene decreased elevated serum creatinine, blood urea nitrogen, renal malondialdehyde and immunoexpression of the proapoptotic protein (Bax), autophagic marker protein (LC3/B), and inducible nitric oxide synthase (iNOS) induced by gentamicin.	rosmarinic acid	18-33	BCL2 associated X, apoptosis regulator	Rattus norvegicus	177-180	
22205146-9	2012	Moreover, RA suppressed the increased ratio of Bax/Bcl-2 at gene level induced by 6-OHDA.	rosmarinic acid	10-12	BCL2 associated X, apoptosis regulator	Rattus norvegicus	47-50	
22205146-10	2012	This indicates that the neurorescue effects of RA against 6-ODHA-induced degeneration of the nigrostriatal dopaminergic system were achieved by decreasing nigral iron levels and regulating the ratio of Bcl-2/Bax gene expression.	rosmarinic acid	47-49	BCL2 associated X, apoptosis regulator	Rattus norvegicus	208-211