PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
17884684-7	2008	The 6-hydroxydopamine-induced decrease in the Bcl-x/Bax ratio was prevented by salvianolic acid B.	salvianolic acid B	79-97	BCL2 associated X, apoptosis regulator	Homo sapiens	52-55	
30204390-10	2016	Conclusion: Sal B is capable of suppressing IHG-induced injury and apoptosis in HUVECs, which might be attributed to the attenuation of oxidative stress, regulation of BCL-2/BAX protein expression, and subsequent suppression of Caspase-3 activity.	salvianolic acid B	12-17	BCL2 associated X, apoptosis regulator	Homo sapiens	174-177	
22252725-5	2012	Furthermore, Sal B down-regulated the HG-mediated Bax expression and AIF nuclear translocation and the release of cytochrome c, but up-regulated the HG-induced BcL-2 expression in SCs.	salvianolic acid B	13-18	BCL2 associated X, apoptosis regulator	Homo sapiens	50-53	
22036624-8	2012	Furthermore, treatment with Sal B down-regulated the IHG-induced release of cytochrome c, AIF nuclear translocation and Bax expression, but mitigated the IHG-mediated down-regulation of BcL-2 expression in SCs.	salvianolic acid B	28-33	BCL2 associated X, apoptosis regulator	Homo sapiens	120-123	
35068334-10	2022	In diabetic thoracic aorta, HG- and CCCP-induced HUVECs, Sal B distinctly increased Bcl-2 expression and reduced BAX, Beclin1, Parkin and Pink1 expression, thereby protecting endothelial cells from apoptosis and mitophagy.	salvianolic acid B	57-62	BCL2 associated X, apoptosis regulator	Homo sapiens	113-116