PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32663610-12	2020	GlcN-induced lipid accumulation was inhibited by the AMPK activator, AICAR.	Glucosamine	0-4	protein kinase AMP-activated catalytic subunit alpha 1	Homo sapiens	53-57	
32663610-13	2020	Phosphorylation of AMPK (p-AMPK) was suppressed by GlcN under NG while increased by GlcN under HG.	Glucosamine	51-55	protein kinase AMP-activated catalytic subunit alpha 1	Homo sapiens	19-23	
32663610-13	2020	Phosphorylation of AMPK (p-AMPK) was suppressed by GlcN under NG while increased by GlcN under HG.	Glucosamine	51-55	protein kinase AMP-activated catalytic subunit alpha 1	Homo sapiens	27-31	
32663610-13	2020	Phosphorylation of AMPK (p-AMPK) was suppressed by GlcN under NG while increased by GlcN under HG.	Glucosamine	84-88	protein kinase AMP-activated catalytic subunit alpha 1	Homo sapiens	19-23	
32663610-13	2020	Phosphorylation of AMPK (p-AMPK) was suppressed by GlcN under NG while increased by GlcN under HG.	Glucosamine	84-88	protein kinase AMP-activated catalytic subunit alpha 1	Homo sapiens	27-31	
32663610-15	2020	Our results collectively suggest that GlcN regulates lipogenesis by sensing the glucose or energy states of normal and excess fuel through AMPK modulation.	Glucosamine	38-42	protein kinase AMP-activated catalytic subunit alpha 1	Homo sapiens	139-143