PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34870709-7	2021	CXCL3 was further identified as an independent prognostic factor for HNSCC patients by Cox regression analysis, and GSEA exhibited that several signaling pathways including Apoptosis, Toll-like receptor, Nod-like receptor, Jak-STAT, and MAPK signaling pathways may be involved in the tumorigenesis of HNSCC.	gsea	116-120	signal transducer and activator of transcription 3	Homo sapiens	227-231	
33820781-9	2021	The GSEA showed that high SUVmax is associated with increased gene expression of inflammatory pathways, including JAK/STAT3 signaling.	gsea	4-8	signal transducer and activator of transcription 3	Homo sapiens	118-123	
34471635-9	2021	GSEA analysis revealed that suppressed expression levels of STAT3 were associated with upregulated oxidative phosphorylation pathways in the neurological injury group of preterm infants.	gsea	0-4	signal transducer and activator of transcription 3	Homo sapiens	60-65	
34148127-14	2021	GSEA revealed that epithelial-mesenchymal transition, IL-6/JAK/STAT3 signaling, the inflammatory response, and TNF-alpha signaling via the NFkappaB pathway were remarkably suppressed pathways in patients with BRAF mutations.	gsea	0-4	signal transducer and activator of transcription 3	Homo sapiens	63-68	
34712264-14	2021	Moreover, GSEA results indicated that the IL6/JAK/STAT3/SIGNALING pathway could be considered as a potential mechanism of genomic instability to influence tumor progression.	gsea	10-14	signal transducer and activator of transcription 3	Homo sapiens	50-55	
35014680-8	2022	GSEA identified the IL-6/JAK/STAT3 pathway as a potential downstream signalling pathway of TGM2.	gsea	0-4	signal transducer and activator of transcription 3	Homo sapiens	29-34	
35609387-15	2022	Protein-protein interaction and GSEA results showed that AU might exert anti-inflammatory effects mainly through the STAT3/NF-kappaB signal pathway.	gsea	32-36	signal transducer and activator of transcription 3	Homo sapiens	117-122	
35205125-7	2022	Moreover, GSEA results showed that immune-related pathways, such as epithelial-mesenchymal transition and IL6/JAK/STAT3 signaling were enriched in the high ARPS risk groups, while the low ARPS risk group mainly regulated metabolism-related processes, such as adipogenesis and bile acid metabolism.	gsea	10-14	signal transducer and activator of transcription 3	Homo sapiens	114-119	
35113040-10	2022	Through GSEA, JAK-STAT is a significantly correlated enrichment pathway.	gsea	8-12	signal transducer and activator of transcription 3	Homo sapiens	18-22	
32686362-11	2020	Finally, GSEA showed that several immune-related gene sets, including IL2/STAT5 signaling, inflammatory response, IL6/JAK/STAT3 signaling, interferon-gamma response and allograft rejection, were elevated in the high-immunity subtype.	gsea	9-13	signal transducer and activator of transcription 3	Homo sapiens	122-127	
33891717-16	2021	GSEA showed that these genes are mainly related to "inflammatory response", "complement", "interferon-alpha response", "IL6/JAK/STAT3 signaling", "TGF-beta signaling", "IL2/STAT5 signaling" and "TNF-alpha signaling via NF-kappaB".	gsea	0-4	signal transducer and activator of transcription 3	Homo sapiens	128-133	
33790966-13	2021	GSEA showed that interleukin-6 (IL-6)/Janus kinase (JAK)/signal transducer and activator of transcription (STAT3) signaling, interferon gamma (IFN-gamma) response, angiogenesis, and coagulation were more highly enriched in the high-risk group and that oxidative phosphorylation was more highly enriched in the low-risk group.	gsea	0-4	signal transducer and activator of transcription 3	Homo sapiens	57-113	
33194434-16	2020	GSEA analysis showed that several gene sets associated with tumor development and metastasis, including TGF-beta signaling, PI3K-AKT-mTOR signaling, and IL6-JAK-STAT3 signaling, were significantly enriched in POLR3G high expression phenotype.	gsea	0-4	signal transducer and activator of transcription 3	Homo sapiens	161-166	
33046027-10	2020	The GSEA results provided further functional annotations, including complement system, IL6/JAK/STAT3 signalling pathway and inflammatory response pathways.	gsea	4-8	signal transducer and activator of transcription 3	Homo sapiens	95-100	
29414823-9	2018	Additionally, Gene Set Enrichment Analysis (GSEA) identified that the JAK/STAT signaling pathway was positively correlated with STK35 expression.	gsea	44-48	signal transducer and activator of transcription 3	Homo sapiens	74-78	
32337262-8	2020	In addition, by GSEA, expression of CXCL1, CXCL11, CXCL2, and CXCL3 was correlated with several signaling pathways, including NOD-like receptor, oxidative phosphorylation, mTORC1, interferon-gamma response, and IL6/JAK/STAT3 pathways.	gsea	16-20	signal transducer and activator of transcription 3	Homo sapiens	219-224	
27824903-11	2016	GSEA indicated that Notch signaling and JAK/STAT3 signaling were significantly associated with the CC/CG genotype (p = 0.004 and p = 0.023, respectively).	gsea	0-4	signal transducer and activator of transcription 3	Homo sapiens	44-49