PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 28105253-9 2016 The reduction in the generation of nitric oxide (NO) and expression of eNOS phosphorylation (Ser1177) in human umbilical vein endothelial cells caused by angiotensin II (Ang II) were restored by DMC or celecoxib. Celecoxib 202-211 angiotensinogen Homo sapiens 154-168 28344559-9 2017 Cyclooxygenase-2 inhibition with celecoxib partially limited the angiotensin II-mediated increases in markers of oxidative stress in humans, offering a potential physiological pathway for the improved cardiovascular risk profile of this drug. Celecoxib 33-42 angiotensinogen Homo sapiens 65-79 28105253-9 2016 The reduction in the generation of nitric oxide (NO) and expression of eNOS phosphorylation (Ser1177) in human umbilical vein endothelial cells caused by angiotensin II (Ang II) were restored by DMC or celecoxib. Celecoxib 202-211 angiotensinogen Homo sapiens 170-176