PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34217798-6	2021	In order to modulate endocannabinoid system, number of agents have been reported amongst which are inhibitors of the monoacylglycerol (MAGL) and fatty acid amide hydrolase (FAAH), the enzymes that hydrolyses 2-AG and AEA respectively.	aea	217-220	monoglyceride lipase	Homo sapiens	135-139	
33834617-2	2021	Both FAAH and MAGL are the endocannabinoid degrading enzymes that hydrolyze several endogenous ligands, mainly anandamide (AEA) and 2-arachidonic glycerol (2-AG), which regulated the various pathophysiological condition of the body like emotion, cognition, energy balance, pain sensation, neuro-inflammation, and cancer cell proliferation.	aea	123-126	monoglyceride lipase	Homo sapiens	14-18	
33834617-3	2021	FAAH and MAGL inhibitors block the metabolism of AEA and 2-AG, and increases endogenous levels of fatty acid amides, and exert various therapeutic effects including chronic pain, metabolic disorders, psychoses, nausea and vomiting, depression, and anxiety disorders, etc.	aea	49-52	monoglyceride lipase	Homo sapiens	9-13	
27516570-12	2016	The main enzymes responsible for the hydrolysis of AEA and 2-AG are FAAH and MAGL, respectively.	aea	51-54	monoglyceride lipase	Homo sapiens	77-81	
16548785-8	2006	The targets include the anandamide-hydrolysing enzyme fatty acid amide hydrolase, the carrier-mediated anandamide transport system and 2-arachidonoyl glycerol-deactivating enzyme monoacylglycerol lipase.	aea	24-34	monoglyceride lipase	Homo sapiens	179-202