PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32797768-5	2020	From the molecular mechanism, it was evident that oxycodone not only decreased the expression levels of Bax, active-caspase 3 protein but also increased the expression levels of Bcl2, p-PI3K, and p-Akt protein in heart tissue of the I/R rats.	Oxycodone	50-59	BCL2, apoptosis regulator	Rattus norvegicus	178-182	
31276638-5	2019	Possible mechanisms might involve the regulatory effect of oxycodone on the MAPK-Bcl2/Bax-caspase-9-caspase-3 pathway, as well as its inhibitory effect on cellular reactive oxygen species (ROS) production and mitochondrial membrane potential activation.	Oxycodone	59-68	BCL2, apoptosis regulator	Rattus norvegicus	81-85	
32572934-7	2020	Compared with the model group, oxycodone group and inhibitor group had an evidently reduced positive expression of BAX (p<0.05) and an evidently raised positive expression of Bcl-2 (p<0.05).	Oxycodone	31-40	BCL2, apoptosis regulator	Rattus norvegicus	175-180	
32572934-13	2020	Compared with the model group, oxycodone group, and inhibitor group had an evidently reduced mRNA expression of BAX (p<0.05) and a significantly raised mRNA expression of Bcl-2 (p<0.05).	Oxycodone	31-40	BCL2, apoptosis regulator	Rattus norvegicus	171-176