PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
18264122-7	2008	CONCLUSIONS AND IMPLICATIONS: There was over-activity of the hexosamine pathway in MCGT1 cells, which may explain the higher expression of TGF-beta1 and p21, the cellular hypertrophy and the increased expression of extracellular matrix (ECM) components in the cells.	Hexosamines	61-71	transforming growth factor, beta 1	Rattus norvegicus	139-148	
18264122-8	2008	By inhibiting the increased activity the hexosamine pathway, rhein decreased TGF-beta1 and p21 expression and thus contributed to the decreased cellular hypertrophy and ECM synthesis.	Hexosamines	41-51	transforming growth factor, beta 1	Rattus norvegicus	77-86	
14559714-5	2004	In this study, we investigated the hypothesis that the effects of glucose on MES matrix production occur via hexosamine regulation of TGF-beta1.	Hexosamines	109-119	transforming growth factor, beta 1	Rattus norvegicus	134-143	
32039769-0	2020	Transcriptional Regulation of Transforming Growth Factor beta1 by Glucose: Investigation into the Role of the Hexosamine Biosynthesis Pathway.	Hexosamines	110-120	transforming growth factor, beta 1	Rattus norvegicus	30-62