PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 10736425-7 2000 Moreover, using IAAQ, we showed that molar excesses of chloroquine, quinine, quinidine, and MK-571 inhibit the photoaffinity labeling of MRP. verlukast 92-98 ATP binding cassette subfamily C member 3 Homo sapiens 137-140 7961706-9 1994 In addition, MK 571 preferentially inhibited photoaffinity labeling of the 190-kDa protein in the MRP transfectants. verlukast 13-19 ATP binding cassette subfamily C member 3 Homo sapiens 98-101 23994649-4 2013 The extracellular levels of PGE2, PGF2alpha, and TXB2 (a metabolite of TXA2) were decreased by treatment with MRP inhibitors (dipyridamole, MK571, and probenecid). verlukast 140-145 ATP binding cassette subfamily C member 3 Homo sapiens 110-113 19732815-4 2010 MK-571, a multidrug resistance associated protein (MRP) inhibitor, significantly suppressed DOX efflux from M5076 ovarian sarcoma cells. verlukast 0-6 ATP binding cassette subfamily C member 3 Homo sapiens 10-49 19732815-4 2010 MK-571, a multidrug resistance associated protein (MRP) inhibitor, significantly suppressed DOX efflux from M5076 ovarian sarcoma cells. verlukast 0-6 ATP binding cassette subfamily C member 3 Homo sapiens 51-54 15210835-4 2004 Calcein efflux was sensitive to temperature, energy depletion, and the MRP antagonist MK571 [3-[[3-[2-(7-chloroquinolin-2-yl)vinyl]phenyl]-(2-dimethylcarbamoylethylsulfanyl)methylsulfanyl] propionic acid]. verlukast 86-91 ATP binding cassette subfamily C member 3 Homo sapiens 71-74 12048155-4 2002 BSO and MK-571, which inhibit the efflux of drugs mediated by multidrug resistance-associated protein (MRP), increased the action of both inhibitors, indicating that the latter enter the cells and that their export is mediated by MRP in both cell lines. verlukast 8-14 ATP binding cassette subfamily C member 3 Homo sapiens 62-101 12048155-4 2002 BSO and MK-571, which inhibit the efflux of drugs mediated by multidrug resistance-associated protein (MRP), increased the action of both inhibitors, indicating that the latter enter the cells and that their export is mediated by MRP in both cell lines. verlukast 8-14 ATP binding cassette subfamily C member 3 Homo sapiens 103-106 12048155-4 2002 BSO and MK-571, which inhibit the efflux of drugs mediated by multidrug resistance-associated protein (MRP), increased the action of both inhibitors, indicating that the latter enter the cells and that their export is mediated by MRP in both cell lines. verlukast 8-14 ATP binding cassette subfamily C member 3 Homo sapiens 230-233 22982073-8 2012 MK-571, chemical inhibitor of MRP2, MRP3, and MRP4, significantly reduced the efflux of glucuronide in the apical-to-basolateral (A-B) and B-A directions in a dose-dependent manner. verlukast 0-6 ATP binding cassette subfamily C member 3 Homo sapiens 36-40 18396325-3 2008 We quantified effects of PSC833, a specific inhibitor of mammalian P-gp (P-glycoprotein, ABCB1), and MK571, which blocks MRP (Multidrug resistance associated protein, ABCC) type transporters, on calcein-am efflux in gill tissue of Mytilus californianus. verlukast 101-106 ATP binding cassette subfamily C member 3 Homo sapiens 121-124 18396325-3 2008 We quantified effects of PSC833, a specific inhibitor of mammalian P-gp (P-glycoprotein, ABCB1), and MK571, which blocks MRP (Multidrug resistance associated protein, ABCC) type transporters, on calcein-am efflux in gill tissue of Mytilus californianus. verlukast 101-106 ATP binding cassette subfamily C member 3 Homo sapiens 126-165 17287390-9 2007 On the other hand, the intracellular resveratrol retention in cells treated with MK571 (3-[[3-[2-(7-chloroquinolin-2-yl)vinyl]phenyl]-(2-dimethylcarbamoylethylsulfanyl)methylsulfanyl] propionic acid), a multidrug resistance-associated protein inhibitor, strongly suggests the involvement of this ABC transporter family in the efflux of resveratrol conjugates from human liver. verlukast 81-86 ATP binding cassette subfamily C member 3 Homo sapiens 203-242 15793098-9 2005 This efflux was decreased by multidrug resistance-associated protein (MRP) inhibitors (probenecid, MK-571) and by ATP depletion. verlukast 99-105 ATP binding cassette subfamily C member 3 Homo sapiens 29-68 15793098-9 2005 This efflux was decreased by multidrug resistance-associated protein (MRP) inhibitors (probenecid, MK-571) and by ATP depletion. verlukast 99-105 ATP binding cassette subfamily C member 3 Homo sapiens 70-73 11212217-8 2001 Moreover, MK571, an inhibitor of MRP, significantly stimulated dye accumulation, whereas inhibitors of the multidrug resistance gene (MDR1) product Pglycoprotein, cyclosporin A and verapamil, did not. verlukast 10-15 ATP binding cassette subfamily C member 3 Homo sapiens 33-36 10421622-11 1999 Efflux of GS-MF was apparently inhibited by MK571, a specific inhibitor for MRP. verlukast 44-49 ATP binding cassette subfamily C member 3 Homo sapiens 76-79