PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23563392-5	2013	However, PGS significantly inhibited the excessive production of NO, PGE2 and pro-inflammatory cytokines, including interleukin-1beta (IL-1beta) and tumor necrosis factor-alpha (TNF-alpha) in a concentration-dependent manner without causing any cytotoxic effects.	Phosphatidylglycerols	9-12	tumor necrosis factor	Mus musculus	149-176	
23563392-5	2013	However, PGS significantly inhibited the excessive production of NO, PGE2 and pro-inflammatory cytokines, including interleukin-1beta (IL-1beta) and tumor necrosis factor-alpha (TNF-alpha) in a concentration-dependent manner without causing any cytotoxic effects.	Phosphatidylglycerols	9-12	tumor necrosis factor	Mus musculus	178-187	
15722559-6	2005	This failure of endogenous PGs to suppress TNF-alpha secretion was explained by the absence of the prostaglandin D2 receptor and the low levels of PGE2 produced during the first 2 h of the LPS response.	Phosphatidylglycerols	27-30	tumor necrosis factor	Mus musculus	43-52	
15722559-7	2005	These studies demonstrate that autocrine regulation of TNF-alpha secretion in response to LPS is greatly facilitated by a COX-1-mediated rapid accumulation of PGs as well by a correspondence between the PGs produced and the receptors expressed by the cells.	Phosphatidylglycerols	159-162	tumor necrosis factor	Mus musculus	55-64	
31669855-3	2020	Thus, we prepared small-sized PS- and PG-loaded liposomes exhibiting narrow distribution, and additionally studied the impact of liposome-pegylation on the reduction of the TNFalpha-production caused by the PS- and PG-liposomes.	Phosphatidylglycerols	215-217	tumor necrosis factor	Mus musculus	173-181	
31669855-7	2020	PG-liposomes always decreased the TNFalpha-levels more potently than PS-liposomes.	Phosphatidylglycerols	0-2	tumor necrosis factor	Mus musculus	34-42	
15181007-8	2004	These results demonstrate autocrine regulation of TNF-alpha secretion by endogenous PGs synthesized primarily by COX-1 in RPM and suggest that COX-1 may play a significant role in the regulation of the early response to endotoxemia.	Phosphatidylglycerols	84-87	tumor necrosis factor	Mus musculus	50-59