PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
9575890-1	1998	The inflammatory cytokine interleukin-1 beta (IL-1 beta) induces both cyclooxygenase-2 (Cox-2) and the inducible nitric oxide synthase (iNOS) with concomitant release of PGs and nitric oxide (NO) by glomerular mesangial cells.	Phosphatidylglycerols	170-173	interleukin 1 beta	Homo sapiens	26-44	
10207838-3	1999	These data showed that endogenous PGs generated by HGF stimulated with IL-1 beta downregulated ICAM-1 expression.	Phosphatidylglycerols	34-37	interleukin 1 beta	Homo sapiens	71-80	
9575890-1	1998	The inflammatory cytokine interleukin-1 beta (IL-1 beta) induces both cyclooxygenase-2 (Cox-2) and the inducible nitric oxide synthase (iNOS) with concomitant release of PGs and nitric oxide (NO) by glomerular mesangial cells.	Phosphatidylglycerols	170-173	interleukin 1 beta	Homo sapiens	46-55	
8118432-6	1993	Cellulose acetate plate electrophoresis of these [35SO4]PG fractions shows that the distribution of PGs alters after treatment with IL-1 beta.	Phosphatidylglycerols	100-103	interleukin 1 beta	Homo sapiens	132-141	
8118432-7	1993	These results indicate that burn wound healing abnormalities (scarring) is related to a change in the level of PGs, and may be modified by IL-1 beta treatment.	Phosphatidylglycerols	111-114	interleukin 1 beta	Homo sapiens	139-148	
7824084-11	1994	Thus, indomethacin was used to determine whether PGs are mediators of the ACTH response to IL-1 beta delivered into the ME.	Phosphatidylglycerols	49-52	interleukin 1 beta	Homo sapiens	91-100	
25465897-5	2014	Finally, p38 mitogen-activated protein kinase (MAPK) inhibitor SB203580 and stress-activated protein kinase/c-Jun N-terminal kinase (SAP/JNK) inhibitor SP600125 were used to pick out the pathway that mediated the IL-1beta-modulated PGs synthesis and gene expression of UGDH, Sp1, Sp3 and c-Krox.	Phosphatidylglycerols	232-235	interleukin 1 beta	Homo sapiens	213-221