PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 6255971-2 1980 In such experimental conditions the Authors could evidence that th aldosterone-inductor effect of angiotensin II is not mediated by cAMP but by PGs and Ca++. Phosphatidylglycerols 144-147 angiotensinogen Homo sapiens 98-112 16204762-8 2005 These results suggest that both an ACE inhibitor and AT1 antagonist (in the presence of angiotensin II) increase duodenal HCO3- secretion via a common pathway, involving bradykinin, NO and PGs. Phosphatidylglycerols 189-192 angiotensinogen Homo sapiens 88-102 12099547-13 2002 There was slightly more increase in Ang II-induced vasoconstrictions in the presence of both NO and PG inhibitors, suggesting that NO may cause release of PGs from these vessels. Phosphatidylglycerols 155-158 angiotensinogen Homo sapiens 36-42 12099547-14 2002 This study demonstrates that the porcine retinal arterioles have the ability to regulate vasoconstriction responses induced by Ang II by synthesis and release of endogenous vasodilating PGs and NO (especially NO); and these substances may play a vital role in porcine retinal circulation. Phosphatidylglycerols 186-189 angiotensinogen Homo sapiens 127-133