PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
16204762-8	2005	These results suggest that both an ACE inhibitor and AT1 antagonist (in the presence of angiotensin II) increase duodenal HCO3- secretion via a common pathway, involving bradykinin, NO and PGs.	Phosphatidylglycerols	189-192	angiotensinogen	Homo sapiens	88-102	
6255971-2	1980	In such experimental conditions the Authors could evidence that th aldosterone-inductor effect of angiotensin II is not mediated by cAMP but by PGs and Ca++.	Phosphatidylglycerols	144-147	angiotensinogen	Homo sapiens	98-112	
12099547-13	2002	There was slightly more increase in Ang II-induced vasoconstrictions in the presence of both NO and PG inhibitors, suggesting that NO may cause release of PGs from these vessels.	Phosphatidylglycerols	155-158	angiotensinogen	Homo sapiens	36-42	
12099547-14	2002	This study demonstrates that the porcine retinal arterioles have the ability to regulate vasoconstriction responses induced by Ang II by synthesis and release of endogenous vasodilating PGs and NO (especially NO); and these substances may play a vital role in porcine retinal circulation.	Phosphatidylglycerols	186-189	angiotensinogen	Homo sapiens	127-133