PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
2420184-3	1986	The deposition of iC3b within the walls of arterioles appears to be due to slow spontaneous activation of the alternative complement pathway and random binding of metastable C3b to proximate hyaluronic acid within the arteriolar wall.	Hyaluronic Acid	191-206	endogenous retrovirus group K member 3	Homo sapiens	19-22	
2420184-4	1986	Since hyaluronic acid does not activate the alternative complement pathway, bound C3b is rapidly inactivated by factors I and H to iC3b, which, along with factor H, remains bound to hyaluronic acid.	Hyaluronic Acid	6-21	endogenous retrovirus group K member 3	Homo sapiens	82-85	
2420184-4	1986	Since hyaluronic acid does not activate the alternative complement pathway, bound C3b is rapidly inactivated by factors I and H to iC3b, which, along with factor H, remains bound to hyaluronic acid.	Hyaluronic Acid	182-197	endogenous retrovirus group K member 3	Homo sapiens	82-85