PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
20164374-8	2010	Interestingly, the treatment of diabetic rats with desferoxamine or tempol (antioxidants/hydroxyl radical scavengers) significantly attenuated the increase in both hydroxyl radical production and in LPO produced by hyperglycemia, preventing apoptosis by reduction of mitochondrial BAX and cytosolic cytochrome c levels.	tempol	68-74	BCL2 associated X, apoptosis regulator	Rattus norvegicus	281-284	
31301730-6	2019	Moreover, tempol inhibited IH-induced apoptosis in pancreatic tissue as evidenced by upregulated Bcl-2 level, and downregulated Bax and cleaved caspase-3 levels.	tempol	10-16	BCL2 associated X, apoptosis regulator	Rattus norvegicus	128-131	
26100649-11	2015	The results demonstrated that compared with the infrasound exposure group, the expression of Bcl-2 was up-regulated and the expressions of Bax and caspase-3 were down-regulated in rats pretreated with NRbt (40 mg/kg) or tempol (40 mg/kg).	tempol	220-226	BCL2 associated X, apoptosis regulator	Rattus norvegicus	139-142	
15833798-8	2005	Superoxide scavenging by Tempol treatment inhibited both the Bax-positive index as well as the apoptotic index of medial SMC in response to vascular injury.	tempol	25-31	BCL2 associated X, apoptosis regulator	Rattus norvegicus	61-64