PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
16670527-6	2006	RESULTS: Using a model of acute trinitrobenzenesulfonic acid (TNBS)-induced colitis, we found that IL-17 was produced in colon tissue at 24 and 48 hours and that IL-17R knockout mice were significantly protected against TNBS-induced weight loss, IL-6 production, colonic inflammation, and local macrophage inflammatory protein-2 induction.	Trinitrobenzenesulfonic Acid	62-66	interleukin 17 receptor A	Mus musculus	162-168	
16670527-6	2006	RESULTS: Using a model of acute trinitrobenzenesulfonic acid (TNBS)-induced colitis, we found that IL-17 was produced in colon tissue at 24 and 48 hours and that IL-17R knockout mice were significantly protected against TNBS-induced weight loss, IL-6 production, colonic inflammation, and local macrophage inflammatory protein-2 induction.	Trinitrobenzenesulfonic Acid	32-60	interleukin 17 receptor A	Mus musculus	162-168	
16670527-6	2006	RESULTS: Using a model of acute trinitrobenzenesulfonic acid (TNBS)-induced colitis, we found that IL-17 was produced in colon tissue at 24 and 48 hours and that IL-17R knockout mice were significantly protected against TNBS-induced weight loss, IL-6 production, colonic inflammation, and local macrophage inflammatory protein-2 induction.	Trinitrobenzenesulfonic Acid	220-224	interleukin 17 receptor A	Mus musculus	162-168	
16670527-9	2006	Furthermore, overexpression of an IL-17R IgG1 fusion protein significantly attenuated colonic inflammation after acute TNBS.	Trinitrobenzenesulfonic Acid	119-123	interleukin 17 receptor A	Mus musculus	34-40