PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
8627184-2	1996	We therefore investigated the in vivo relevance of this interaction in an experimental model for a Th1-mediated disease, the hapten reagent (2,4,6-trinitrobenzene sulfonic acid [TNBS])-induced colitis.	Trinitrobenzenesulfonic Acid	178-182	negative elongation factor complex member C/D	Homo sapiens	99-102	
25177159-6	2014	TNBS elicits Th-1 driven immune response, whereas oxazolone predominantly exhibits immune response of Th-2 phenotype.	Trinitrobenzenesulfonic Acid	0-4	negative elongation factor complex member C/D	Homo sapiens	13-17	
12901851-9	2003	Pretreatment with rhG-CSF drastically attenuated the degree of TNBS-induced colitis through selective downregulation of Th1-associated cytokines.	Trinitrobenzenesulfonic Acid	63-67	negative elongation factor complex member C/D	Homo sapiens	120-123	
11207312-1	2001	Trinitrobenzene sulfonic acid (TNBS)-induced colitis is an IL-12-driven, Th1 T cell-mediated colitis that resembles human Crohn"s disease.	Trinitrobenzenesulfonic Acid	0-29	negative elongation factor complex member C/D	Homo sapiens	73-76	
11207312-1	2001	Trinitrobenzene sulfonic acid (TNBS)-induced colitis is an IL-12-driven, Th1 T cell-mediated colitis that resembles human Crohn"s disease.	Trinitrobenzenesulfonic Acid	31-35	negative elongation factor complex member C/D	Homo sapiens	73-76	
27206705-2	2016	The aim of this study was to analyze whether and how low doses of haptens, by entry through the skin or gastrointestinal tract, affect the outcome of the predominantly Th1/Th17-mediated 2,4,6-trinitro-benzenesulfonic acid-induced colitis, which mimics an autoimmune bowl disease in man.	Trinitrobenzenesulfonic Acid	186-221	negative elongation factor complex member C/D	Homo sapiens	168-171