PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 26235132-7 2015 Moreover, the RNA interference (RNAi) of NFkappaB suppressed the expression of iNOS induced by lipid A. Lipid A 95-102 nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105 Mus musculus 41-49 28587938-11 2017 Furthermore, etidronate inhibited the translocation of NF-kappaB but not p38 MAPK in J774.1 cells stimulated with Pam3CSK4 or lipid A. Lipid A 126-133 nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105 Mus musculus 55-64 26235132-8 2015 We demonstrated that lipid A-induced iNOS expression in ECPC-4 cells was mainly regulated by the activation of NFkappaB-IkappaB intracellular signaling pathway. Lipid A 21-28 nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105 Mus musculus 111-119 10064907-9 1999 LPS stimulation of p38 phosphorylation, NF-kappaB activation, and surface IgM expression were all blocked completely by lipid A-like endotoxin antagonists whether or not CD14 was present. Lipid A 120-127 nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105 Mus musculus 40-49 24686069-6 2014 We also demonstrated that lptA mutant bacteria were more susceptible to human and murine cathelicidins due to increased binding by these peptides and that the differential induction of NF-kappaB by wild-type and unmodified lipid A was more pronounced in the presence of CAMPs. Lipid A 223-230 nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105 Mus musculus 185-194 25837580-7 2015 The afterglow exposure also results in a reduction of the lipid A proinflammatory activity, assessed by the net decrease of the redox-sensitive NFkappaB transcription factor nuclear translocation in murine aortic endothelial cells stimulated with control vs afterglow-treated (pure and extracted) lipid A. Lipid A 58-65 nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105 Mus musculus 144-152 21152039-0 2010 ATF3 plays a key role in Kdo2-lipid A-induced TLR4-dependent gene expression via NF-kappaB activation. Lipid A 30-37 nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105 Mus musculus 81-90 14529784-2 2003 This study examines whether lipid A may directly modulate the radioresponse of tumor cells by activating inducible nitric oxide synthase (iNOS) or cyclooxygenase-2 (COX-2) through nuclear factor-kappaB (NF-kappaB) signaling. Lipid A 28-35 nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105 Mus musculus 180-201 14529784-2 2003 This study examines whether lipid A may directly modulate the radioresponse of tumor cells by activating inducible nitric oxide synthase (iNOS) or cyclooxygenase-2 (COX-2) through nuclear factor-kappaB (NF-kappaB) signaling. Lipid A 28-35 nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105 Mus musculus 203-212 12717385-8 2003 Lipid A induces NF-kappaB activation in HSCs from TLR4-sufficient (C3H/OuJ) mice but not from TLR4-deficient (C3H/HeJ) mice. Lipid A 0-7 nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105 Mus musculus 16-25 10064907-11 1999 Our data support the hypothesis that lipid A-mediated activation of cells requires stimulation of a putative lipid A sensor that is downstream of CD14, but upstream of p38 and NF-kappaB. Lipid A 37-44 nuclear factor of kappa light polypeptide gene enhancer in B cells 1, p105 Mus musculus 176-185