PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33413231-8	2021	Moreover, the data indicated that both the ER-resident and the secreted AGR2 enhance the survival capacity of PANC-1 cells after tunicamycin-induced ER stress and gemcitabine treatment.	Tunicamycin	129-140	anterior gradient 2, protein disulphide isomerase family member	Homo sapiens	72-76	
33822017-9	2021	In HT-29 cells, tunicamycin-induced ER stress triggered AGR2 intracellular expression and its secretion.	Tunicamycin	16-27	anterior gradient 2, protein disulphide isomerase family member	Homo sapiens	56-60	
27941872-7	2017	In vitro, AGR2 expression was stimulated by tunicamycin-induced endoplasmic reticulum (ER) stress in both KRAS wild-type normal pancreas cells, as well as in KRAS mutated pancreatic cancer cells and was essential for ER homoeostasis.	Tunicamycin	44-55	anterior gradient 2, protein disulphide isomerase family member	Homo sapiens	10-14	
34237462-12	2021	The mutant AGR2 showed reduced capacity to bind MUC2 and alleviate tunicamycin-induced ER stress.	Tunicamycin	67-78	anterior gradient 2, protein disulphide isomerase family member	Homo sapiens	11-15