PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 1376736-7 1992 Pretreatment of basophils with lactic acid led to abrogation of sensitivity to HRF. Lactic Acid 31-42 tumor protein, translationally-controlled 1 Homo sapiens 79-82 1695233-9 1990 Stripping of the membrane-bound IgE by incubating basophils with lactic acid causes a significant loss of sensitivity to HRF generated by PBMCs, T cells, B cells, and macrophages, as well as to HRF recovered from bronchoalveolar fluid. Lactic Acid 65-76 tumor protein, translationally-controlled 1 Homo sapiens 121-124 1695233-9 1990 Stripping of the membrane-bound IgE by incubating basophils with lactic acid causes a significant loss of sensitivity to HRF generated by PBMCs, T cells, B cells, and macrophages, as well as to HRF recovered from bronchoalveolar fluid. Lactic Acid 65-76 tumor protein, translationally-controlled 1 Homo sapiens 194-197 2439589-7 1987 Histamine release occurred via a mechanism which is IgE-dependent in that: basophils desensitized by exposure to anti-IgE in the absence of calcium no longer respond to HRF, and desensitization with HRF reduces responsiveness to anti-IgE; and removal of IgE from the basophil surface by using lactic acid renders cells unresponsive to HRF. Lactic Acid 293-304 tumor protein, translationally-controlled 1 Homo sapiens 199-202 2439589-7 1987 Histamine release occurred via a mechanism which is IgE-dependent in that: basophils desensitized by exposure to anti-IgE in the absence of calcium no longer respond to HRF, and desensitization with HRF reduces responsiveness to anti-IgE; and removal of IgE from the basophil surface by using lactic acid renders cells unresponsive to HRF. Lactic Acid 293-304 tumor protein, translationally-controlled 1 Homo sapiens 199-202