PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
1933853-0	1991	Nitrosamine-induced cancer: selective repair and conformational differences between O6-methylguanine residues in different positions in and around codon 12 of rat H-ras.	O-(6)-methylguanine	84-100	HRas proto-oncogene, GTPase	Rattus norvegicus	163-168	
1807448-0	1991	Positional effects on the structure and stability of abbreviated H-ras DNA sequences containing O6-methylguanine residues at codon 12.	O-(6)-methylguanine	96-112	HRas proto-oncogene, GTPase	Rattus norvegicus	65-70	
1807448-1	1991	Activation of the H-ras protooncogene in rats by methylating carcinogens results from a G-to-A transition mutation at the second position of codon 12 (GGA), presumably due to formation of an O6-methylguanine (m6G) at this position.	O-(6)-methylguanine	191-207	HRas proto-oncogene, GTPase	Rattus norvegicus	18-23	
10767621-9	2000	O(6)-methylguanines are repaired at a similar slow rate in both transcriptionally active (H-ras, beta-actin) and inactive genes (IgE heavy chain; bulk DNA) of the target mammary epithelia (which express the repair protein O(6)-alkylguanine-DNA alkyltransferase (AGT) at a very low level).	O-(6)-methylguanine	1-19	HRas proto-oncogene, GTPase	Rattus norvegicus	90-95	
9465068-4	1998	In mammary epithelia (very low expression of O6-alkylguanine-DNA alkyltransferase, MGMT), O6-methylguanine (O6-MeGua) was eliminated from transcribed (H-ras and beta-actin) and inactive genes (IgE heavy chain) at the same slow rate as determined for bulk genomic DNA.	O-(6)-methylguanine	90-106	HRas proto-oncogene, GTPase	Rattus norvegicus	151-156	
9465068-4	1998	In mammary epithelia (very low expression of O6-alkylguanine-DNA alkyltransferase, MGMT), O6-methylguanine (O6-MeGua) was eliminated from transcribed (H-ras and beta-actin) and inactive genes (IgE heavy chain) at the same slow rate as determined for bulk genomic DNA.	O-(6)-methylguanine	108-116	HRas proto-oncogene, GTPase	Rattus norvegicus	151-156	
9465068-5	1998	The persistence of O6-MeGua in DNA correlated with a high frequency of G:C --> A:T transition mutations at codon 12 of the H-ras gene in MeNU-induced tumors.	O-(6)-methylguanine	19-27	HRas proto-oncogene, GTPase	Rattus norvegicus	126-131	
7688078-0	1993	Nonrandom distribution of O6-methylguanine in H-ras gene sequence from DNA modified with N-methyl-N-nitrosourea.	O-(6)-methylguanine	26-42	HRas proto-oncogene, GTPase	Rattus norvegicus	46-51	
7688078-1	1993	The distribution of O6-meG in the rat H-ras gene sequence was studied using PCR by transition of O6-meG to adenine during the reaction.	O-(6)-methylguanine	20-26	HRas proto-oncogene, GTPase	Rattus norvegicus	38-43	
1933853-5	1991	We have measured accurate rates of repair by the E. coli and gene O6-alkylguanine-DNA-alkyltransferase of an O6-methylguanine in various positions in chemically synthesized 15-base pair DNA duplexes having the H-ras sequence.	O-(6)-methylguanine	109-125	HRas proto-oncogene, GTPase	Rattus norvegicus	210-215	
2643488-0	1989	Excision repair of O6-methylguanine synthesized at the rat H-ras N-methyl-N-nitrosourea activation site and introduced into Escherichia coli.	O-(6)-methylguanine	19-35	HRas proto-oncogene, GTPase	Rattus norvegicus	59-64	
2643488-1	1989	O6-methylguanine (O6-methylG) is believed to be the premutagenic lesion responsible for mutational activation of the H-ras proto-oncogene in rats treated with N-methyl-N-nitrosourea (MNU).	O-(6)-methylguanine	0-16	HRas proto-oncogene, GTPase	Rattus norvegicus	117-122	
2643488-1	1989	O6-methylguanine (O6-methylG) is believed to be the premutagenic lesion responsible for mutational activation of the H-ras proto-oncogene in rats treated with N-methyl-N-nitrosourea (MNU).	O-(6)-methylguanine	18-28	HRas proto-oncogene, GTPase	Rattus norvegicus	117-122