PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
8683108-3	1996	Functional studies using chloramphenicol acetyltransferase reporter gene constructs that contained deleted 5" flanking region junB sequences identified a region located between -194 and -87 that contains an Ets binding site and a putative cAMP response element binding site (CRE-like).	Cyclic AMP	239-243	jun B proto-oncogene	Mus musculus	126-130	
11024538-7	2000	Both the inhibition of the MEKK1/MEK4/JNK pathway, leading to the reduction in phosphorylated c-Jun, and the stimulation of JunB, are mediated through the specific VPAC1 receptor and the cAMP/PKA pathway.	Cyclic AMP	187-191	jun B proto-oncogene	Mus musculus	124-128	
8392062-6	1993	Since cAMP-dependent protein kinase activity of J774 cells was inhibited by H-89, but not by H-7, LPS appears to activate junB through a cascade involving two steps, the one sensitive to H-89 and the other to H-7.	Cyclic AMP	6-10	jun B proto-oncogene	Mus musculus	122-126	
8265655-1	1993	JunB is an immediate early transcription factor that is induced by a variety of extracellular signaling agents, including growth factors, phorbol esters, and agents that elevate cyclic AMP.	Cyclic AMP	178-188	jun B proto-oncogene	Mus musculus	0-4	
8265655-3	1993	By using the JunB gene together with flanking DNA in transfection experiments, we show that a serum response element (SRE) and/or a cAMP response element (CRE) downstream of the gene mediate the response of the gene in mouse NIH 3T3 cells to serum, platelet-derived growth factor, basic fibroblast growth factor, phorbol ester, and forskolin.	Cyclic AMP	132-136	jun B proto-oncogene	Mus musculus	13-17	
1850843-1	1991	Products of the adenovirus E1A gene can act synergistically with cAMP to activate transcription of several viral early genes and the cellular genes c-fos and jun-B.	Cyclic AMP	65-69	jun B proto-oncogene	Mus musculus	158-163	
2559873-4	1989	Cytoplasmic levels of c-fos and junB mRNAs are rapidly increased by cAMP, and the induction is substantially stronger in the presence of E1A protein.	Cyclic AMP	68-72	jun B proto-oncogene	Mus musculus	32-36	
2562123-3	1989	The proto-oncogenes jun B, c-fos, and to a lesser extent jun D were stimulated by increasing the intracellular concentration of cAMP, whereas the TPA stimulation of c-jun and c-myc was inhibited under these conditions.	Cyclic AMP	128-132	jun B proto-oncogene	Mus musculus	20-25