PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
12799645-3	2003	Pancreatic cancer frequently has a dysregulated p21(ras) pathway and therefore appears to be a suitable target for BPs that interfere with the prenylation of small GTP-binding proteins such as p21(ras).	Diphosphonates	115-118	H3 histone pseudogene 16	Homo sapiens	48-51	
12799645-3	2003	Pancreatic cancer frequently has a dysregulated p21(ras) pathway and therefore appears to be a suitable target for BPs that interfere with the prenylation of small GTP-binding proteins such as p21(ras).	Diphosphonates	115-118	H3 histone pseudogene 16	Homo sapiens	193-196	
31423185-8	2019	Consistent with the observed growth inhibitory effects, BPs also inhibited the cell cycle by promoting G1 phase arrest and the downregulation of cyclin D1 and upregulation of p21.	Diphosphonates	56-59	H3 histone pseudogene 16	Homo sapiens	175-178