PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
11976278-6	2002	We conclude that thioridazine interacts with the inner membrane of mitochondria, more likely close to its surface, acquiring antioxidant activity toward processes with potential implications in apoptosis such as O2*- accumulation, as well as LPO, MPT and associated release of cytochrome c.	Thioridazine	17-29	cytochrome c, somatic	Homo sapiens	277-289	
29960166-5	2018	TDZ treatment induced nuclear condensation, mitochondrial membrane potential loss, mitochondrial cytochrome c release, activation of caspase-9 and caspase-3 substantiating mitochondrial pathways of apoptosis in cells.	Thioridazine	0-3	cytochrome c, somatic	Homo sapiens	97-109	
24130798-2	2013	Cytochrome c was irradiated with UV light for 120 min, over a pH range from 4.0 to 8.0, in the absence and in the presence of different concentrations of thioridazine (TR) and fluphenazine (FP).	Thioridazine	154-166	cytochrome c, somatic	Homo sapiens	0-12