PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 32768653-7 2020 Intriguingly, Peli1 upregulation induced by Meth was dependent on TRIF rather than the myloid differentiation factor 88 (MyD88) pathway, since the silencing of TRIF significantly suppressed Meth-induced Peli1 upregulation, while MyD88 knockdown had no obvious impact. Methamphetamine 44-48 myeloid differentiation primary response gene 88 Mus musculus 229-234 32768653-7 2020 Intriguingly, Peli1 upregulation induced by Meth was dependent on TRIF rather than the myloid differentiation factor 88 (MyD88) pathway, since the silencing of TRIF significantly suppressed Meth-induced Peli1 upregulation, while MyD88 knockdown had no obvious impact. Methamphetamine 190-194 myeloid differentiation primary response gene 88 Mus musculus 229-234 32941923-0 2021 Melatonin ameliorates methamphetamine-induced cognitive impairments by inhibiting neuroinflammation via suppression of the TLR4/MyD88/NFkappaB signaling pathway in the mouse hippocampus. Methamphetamine 22-37 myeloid differentiation primary response gene 88 Mus musculus 128-133 32941923-8 2021 METH-induced activation of glial cells is associated with the activation of the TLR4/MyD88/NFkappaB signaling pathway. Methamphetamine 0-4 myeloid differentiation primary response gene 88 Mus musculus 85-90 34381368-0 2021 Antibiotics Attenuate Methamphetamine-Induced Hepatotoxicity by Regulating Oxidative Stress and TLR4/MyD88/Traf6 Axis. Methamphetamine 22-37 myeloid differentiation primary response gene 88 Mus musculus 101-106 34381368-9 2021 Western blot analysis indicated that antibiotics inhibited the increase of TLR4, MyD88 and Traf6 induced by METH. Methamphetamine 108-112 myeloid differentiation primary response gene 88 Mus musculus 81-86 34381368-10 2021 This research suggests that antibiotics may play an important role in preventing METH-induced liver injury by regulating oxidative stress and TLR4/MyD88/Traf6 axis, though further investigation is required. Methamphetamine 81-85 myeloid differentiation primary response gene 88 Mus musculus 147-152 33359793-7 2021 METH up-regulated inflammation-related factors, such as caspase1, TNF-alpha and IL-18, by activating the toll-like receptors 4 (TLR4)/myeloid differentiation factor 88 (Myd88)/nuclear factor kappaB (NF-kappaB) pathway and reduced occludin protein expression. Methamphetamine 0-4 myeloid differentiation primary response gene 88 Mus musculus 169-174