PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33430700-9	2021	In conclusion, our study showed that PA could destroy differentiative capacity of C2C12 myoblasts by affecting the expression of Pax7, MyoD and MyoG, and OA could improve this impairment through PI3K /Akt signaling pathway.	Protactinium	37-39	thymoma viral proto-oncogene 1	Mus musculus	201-204	
34783223-11	2021	Through molecular dynamics simulation, we observed that Pa was bound to the catalytic pocket of Akt and effectively inhibited the biological activity of Akt.	Protactinium	56-58	thymoma viral proto-oncogene 1	Mus musculus	96-99	
34783223-11	2021	Through molecular dynamics simulation, we observed that Pa was bound to the catalytic pocket of Akt and effectively inhibited the biological activity of Akt.	Protactinium	56-58	thymoma viral proto-oncogene 1	Mus musculus	153-156	
34783223-12	2021	These results indicated that Pa significantly relieves LPS-induced ALI by activating the Akt/NF-kappaB signaling pathway.	Protactinium	29-31	thymoma viral proto-oncogene 1	Mus musculus	89-92	
25590691-4	2015	Moreover, the active component of CKS, platyconic acid A (PA), suppressed PMA-induced MUC5AC mRNA expression (from 2.1 +- 0.2 to 1.1 +- 0.1) by inhibiting NF-kappaB activation (from 2.3 +- 0.2 to 1.2 +- 0.1) via Akt (from 3.7 +- 0.3 to 2.1 +- 0.2) (p < 0.01) in A549 cells.	Protactinium	58-60	thymoma viral proto-oncogene 1	Mus musculus	212-215	
25923736-4	2015	Here, we found that PKB-mediated phosphorylation of Thr649 on AS160/TBC1D4 represented one of the major PAS-binding signals in the heart in response to insulin.	Protactinium	104-107	thymoma viral proto-oncogene 1	Mus musculus	20-23