PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23691121-5	2013	Nox inhibitors diphenyleneiodonium chloride (DPI) and Duox1 siRNA were used to investigate whether Duox1 is involved in PA-LPS-induced MUC5AC and Clca3 expression both in vitro and in vivo.	Protactinium	120-122	dual oxidase 1	Mus musculus	99-104	
23691121-3	2013	Our aim was to clarify whether Duox1 was also involved in the PA-LPS-induced MUC5AC and calcium dependent chloride channel 3(Clca3), another recognized marker of goblet cell hyperplasia and mucus hyper-production.	Protactinium	62-64	dual oxidase 1	Mus musculus	31-36	
23691121-8	2013	Knockdown of Duox1 markedly inhibited PA-LPS-induced MUC5AC expression via a ROS-TGF-alpha cascade in A549 cells.	Protactinium	38-40	dual oxidase 1	Mus musculus	13-18	
23691121-10	2013	CONCLUSIONS: We demonstrate for the first time that PA-LPS-induced MUC5AC and Clca3 expression is partly through Duox1, and provide supportive evidence for Duox1 as a potential target in treatments of mucin over-production diseases.	Protactinium	52-54	dual oxidase 1	Mus musculus	113-118	
23691121-10	2013	CONCLUSIONS: We demonstrate for the first time that PA-LPS-induced MUC5AC and Clca3 expression is partly through Duox1, and provide supportive evidence for Duox1 as a potential target in treatments of mucin over-production diseases.	Protactinium	52-54	dual oxidase 1	Mus musculus	156-161