PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23743712-7	2013	Furthermore, treatment of Arabidopsis wild-type seedlings with succinylacetone, an abnormal metabolite caused by loss of FAH in the Tyr degradation pathway, mimicked the sscd1 cell death phenotype.	succinylacetone	63-78	fumarylacetoacetase	Arabidopsis thaliana	121-124	
23743712-7	2013	Furthermore, treatment of Arabidopsis wild-type seedlings with succinylacetone, an abnormal metabolite caused by loss of FAH in the Tyr degradation pathway, mimicked the sscd1 cell death phenotype.	succinylacetone	63-78	fumarylacetoacetase	Arabidopsis thaliana	170-175	
30666736-2	2019	The sscd1 mutant accumulates succinylacetone (SUAC), an abnormal metabolite caused by loss of FAH.	succinylacetone	29-44	fumarylacetoacetase	Arabidopsis thaliana	4-9	
30666736-2	2019	The sscd1 mutant accumulates succinylacetone (SUAC), an abnormal metabolite caused by loss of FAH.	succinylacetone	29-44	fumarylacetoacetase	Arabidopsis thaliana	94-97	
30666736-2	2019	The sscd1 mutant accumulates succinylacetone (SUAC), an abnormal metabolite caused by loss of FAH.	succinylacetone	46-50	fumarylacetoacetase	Arabidopsis thaliana	4-9	
30666736-2	2019	The sscd1 mutant accumulates succinylacetone (SUAC), an abnormal metabolite caused by loss of FAH.	succinylacetone	46-50	fumarylacetoacetase	Arabidopsis thaliana	94-97	
30666736-7	2019	In addition, Arabidopsis WT seedlings treated with SUAC mimic sscd1 in decline of ALAD activity and accumulation of Pchlide as well as cell death.	succinylacetone	51-55	fumarylacetoacetase	Arabidopsis thaliana	62-67	
30666736-8	2019	These results demonstrate that increase in Pchlide causes cell death in sscd1 upon re-illumination and suggest that a decline in the Pchlide pool due to inhibition of ALAD activity by SUAC impairs the repression of ALA synthesis from the light-dark transition by feedback control, resulting in activation of the Chl biosynthesis pathway and accumulation of Pchlide in the dark.	succinylacetone	184-188	fumarylacetoacetase	Arabidopsis thaliana	72-77	
27097641-7	2016	In addition, a high concentration of sugar attenuated cell death of Arabidopsis wild-type seedlings caused by treatment with exogenous succinylacetone, an abnormal metabolite resulting from the loss of FAH in the Tyr degradation pathway.	succinylacetone	135-150	fumarylacetoacetase	Arabidopsis thaliana	202-205	
27097641-8	2016	These results indicated that (1) sugar could suppress cell death in sscd1, which might be because sugar supply enhances the resistance of Arabidopsis seedlings to toxic effects of succinylacetone and reduces the accumulation of Tyr degradation intermediates, resulting in suppression of cell death; and (2) sucrose-processing genes cell-wall invertase 1 and alkaline/neutral invertase G might be involved in the cell death in sscd1.	succinylacetone	180-195	fumarylacetoacetase	Arabidopsis thaliana	68-73