PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32801121-6	2020	In our established human salivary gland-derived organoid culture system, we successfully induced an organoid swelling revealing the function of saliva secretion by the stimulation of carbachol, a non-selective cholinergic agonist, and forskolin, an activator of cystic fibrosis transmembrane conductance regulator (CFTR).	Carbachol	183-192	CF transmembrane conductance regulator	Homo sapiens	262-313	
23744739-6	2013	Carbachol and cAMP redistributed CFTR to the apical membrane.	Carbachol	0-9	CF transmembrane conductance regulator	Homo sapiens	33-37	
32801121-6	2020	In our established human salivary gland-derived organoid culture system, we successfully induced an organoid swelling revealing the function of saliva secretion by the stimulation of carbachol, a non-selective cholinergic agonist, and forskolin, an activator of cystic fibrosis transmembrane conductance regulator (CFTR).	Carbachol	183-192	CF transmembrane conductance regulator	Homo sapiens	315-319	
31048413-3	2019	We report here that secretagogues (agonists that stimulate secretion) such as the peptide hormone vasoactive intestinal peptide (VIP) and muscarinic agonist carbachol increase CFTR aggregation into cholesterol-dependent clusters, reduce CFTR lateral mobility within and between membrane microdomains, and trigger the fusion of clusters into large (3.0 microm2) ceramide-rich platforms.	Carbachol	157-166	CF transmembrane conductance regulator	Homo sapiens	176-180	
31048413-3	2019	We report here that secretagogues (agonists that stimulate secretion) such as the peptide hormone vasoactive intestinal peptide (VIP) and muscarinic agonist carbachol increase CFTR aggregation into cholesterol-dependent clusters, reduce CFTR lateral mobility within and between membrane microdomains, and trigger the fusion of clusters into large (3.0 microm2) ceramide-rich platforms.	Carbachol	157-166	CF transmembrane conductance regulator	Homo sapiens	237-241	
27830759-8	2016	MCC velocity was most dramatically accelerated by the synergistic combination of forskolin and carbachol, which produced near-maximal clearance rates regardless of prior treatment with CFTR or ENaC inhibitors.	Carbachol	95-104	CF transmembrane conductance regulator	Homo sapiens	185-189	
24040112-9	2013	CFTR dependent currents following 18-24 hours of cold storage for forskolin/IBMX, carbachol, and forskolin/IBMX+carbachol stimulation (n=17 non-CF subjects) were 44%, 47.5%, and 47.3%, respectively of those in fresh biopsies.	Carbachol	82-91	CF transmembrane conductance regulator	Homo sapiens	0-4	
23827485-7	2014	With human bronchial rings, we observed that whatever the compound used including salbutamol, the activation of muscular CFTR leads to a bronchodilation after constriction with carbachol.	Carbachol	177-186	CF transmembrane conductance regulator	Homo sapiens	121-125	
24040112-9	2013	CFTR dependent currents following 18-24 hours of cold storage for forskolin/IBMX, carbachol, and forskolin/IBMX+carbachol stimulation (n=17 non-CF subjects) were 44%, 47.5%, and 47.3%, respectively of those in fresh biopsies.	Carbachol	112-121	CF transmembrane conductance regulator	Homo sapiens	0-4	
21030607-8	2011	In contrast, carbachol induced membrane recruitment of NKCC1 and CFTR in all crypt and villus enterocytes, NKCC1 in all goblet cells, and NBCe1 in all villus enterocytes.	Carbachol	13-22	CF transmembrane conductance regulator	Homo sapiens	65-69	
23784542-0	2013	Carbachol-induced MUC17 endocytosis is concomitant with NHE3 internalization and CFTR membrane recruitment in enterocytes.	Carbachol	0-9	CF transmembrane conductance regulator	Homo sapiens	81-85	
23784542-7	2013	CCh stimulation concomitantly internalized the Na(+/)H(+) exchanger 3 (NHE3) and recruited cystic fibrosis transmembrane conductance regulator (CFTR) to the apical membranes, a process that was important for CFTR-mediated bicarbonate secretion necessary for proper gel-forming mucin unfolding.	Carbachol	0-3	CF transmembrane conductance regulator	Homo sapiens	91-142	
23784542-7	2013	CCh stimulation concomitantly internalized the Na(+/)H(+) exchanger 3 (NHE3) and recruited cystic fibrosis transmembrane conductance regulator (CFTR) to the apical membranes, a process that was important for CFTR-mediated bicarbonate secretion necessary for proper gel-forming mucin unfolding.	Carbachol	0-3	CF transmembrane conductance regulator	Homo sapiens	144-148	
23784542-7	2013	CCh stimulation concomitantly internalized the Na(+/)H(+) exchanger 3 (NHE3) and recruited cystic fibrosis transmembrane conductance regulator (CFTR) to the apical membranes, a process that was important for CFTR-mediated bicarbonate secretion necessary for proper gel-forming mucin unfolding.	Carbachol	0-3	CF transmembrane conductance regulator	Homo sapiens	208-212	
20739756-8	2010	First, CFTR-dependent secretion was strongly potentiated by low VIP and carbachol concentrations that individually were unable to stimulate secretion.	Carbachol	72-81	CF transmembrane conductance regulator	Homo sapiens	7-11	
20478977-3	2010	Prostaglandin E(2) (PGE(2))- and carbachol-stimulated mucus release was severely inhibited in the absence of serosal HCO(3)(-), HCO(3)(-) transport, or functional cystic fibrosis transmembrane conductance regulator (CFTR).	Carbachol	33-42	CF transmembrane conductance regulator	Homo sapiens	163-214	
20478977-3	2010	Prostaglandin E(2) (PGE(2))- and carbachol-stimulated mucus release was severely inhibited in the absence of serosal HCO(3)(-), HCO(3)(-) transport, or functional cystic fibrosis transmembrane conductance regulator (CFTR).	Carbachol	33-42	CF transmembrane conductance regulator	Homo sapiens	216-220