PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 18155849-0 2008 Fos expression in pontomedullary catecholaminergic cells following rapid eye movement sleep-like episodes elicited by pontine carbachol in urethane-anesthetized rats. Carbachol 126-135 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 0-3 28966579-7 2017 Inhibition of the MS with muscimol pre-treatment attenuated both carbachol-evoked c-Fos-ir and theta activation. Carbachol 65-74 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 82-87 18155849-5 2008 The percentage of Fos-positive TH cells was negatively correlated with the cumulative duration of REMS-like episodes induced during 140 min prior to brain harvesting in the A7 and rostral A5 groups bilaterally (P < 0.01 for both), and in SubC neurons on the side opposite to carbachol injection (P < 0.05). Carbachol 278-287 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 18-21 13679249-8 2003 Repeated injections of carbachol (30 pmol) into the LSD produced Fos immunoreactivity in the ipsilateral side of the LSV. Carbachol 23-32 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 65-68 11074189-8 2000 Rats with seizures induced by PAG carbachol microinjections exhibited dense c-fos-like immunoreactivity in the dentate gyrus but not the CA(1) or CA(3) regions, amygdala, piriform cortex, perirhinal cortex or hypothalamus. Carbachol 34-43 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 76-81 12370534-0 2002 Regulation of carbachol-induced c-fos mRNA expression in AR42J cells by somatostatin receptor subtypes 1, 2, and 3. Carbachol 14-23 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 32-37 12086906-6 2001 CCh induced time-and dose-dependent increases in the c-fos and c-jun early-response genes, which were blocked by m1 and m3 inhibition but not by m2 inhibition. Carbachol 0-3 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 53-58 12676367-0 2003 Rewarding injections of the cholinergic agonist carbachol into the ventral tegmental area induce locomotion and c-Fos expression in the retrosplenial area and supramammillary nucleus. Carbachol 48-57 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 112-117 12676367-2 2003 To determine what brain regions are activated by such rewarding injections we studied the expression of the transcription factor c-Fos in local and distant brain regions following ventral tegmental injections of carbachol in rats. Carbachol 212-221 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 129-134 12676367-5 2003 Ventral tegmental injections of carbachol induced c-Fos expression throughout the brain. Carbachol 32-41 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 50-55 10404113-1 1999 We used immunocytochemistry to determine the regional and temporal distribution of Fos protein expression in awake and unrestrained rats after a unilateral stereotaxic microinjection of a cholinergic agonist, carbachol, in the thalamic ventroposterolateral and reticular nuclei, previously shown to cause limbic and generalized convulsive seizures. Carbachol 209-218 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 83-86 10585535-4 1999 Supershift analysis revealed that the carbachol-induced AP-1 complex was composed predominantly of Jun D and c-Fos. Carbachol 38-47 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 109-114 10404113-2 1999 The microinjection of carbachol elicits behavioral alterations including immobilization, staring, facial and jaw clonus, rearing, and falling, followed by recurrent generalized convulsive seizures, and a pattern of c-fos expression throughout the brain. Carbachol 22-31 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 215-220 10404113-3 1999 In addition to the hypothalamic paraventricular and supraoptic nuclei, the initial induction of c-fos expression was observed as early as 15 minutes after the carbachol microinjection, in the piriform and entorhinal cortices, the thalamic paraventricular, the supramammilary, the lateral parabrachial nuclei, and the central gray. Carbachol 159-168 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 96-101 9418962-2 1997 Activation of this neurotransmitter receptor by the stable acetylcholine analog carbachol (CCh) triggers transducing events, modulating c-fos expression and cellular proliferation. Carbachol 80-89 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 136-141 9418962-2 1997 Activation of this neurotransmitter receptor by the stable acetylcholine analog carbachol (CCh) triggers transducing events, modulating c-fos expression and cellular proliferation. Carbachol 91-94 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 136-141 9259051-3 1996 We have now found in rat aorta that carbachol, a muscarinic cholinergic agonist that promotes release of nitric oxide (NO), inhibits expression of c-fos and c-jun mRNA induced by alpha 1 receptors. Carbachol 36-45 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 147-152 9259051-4 1996 NO synthase inhibitors blocked the effects of carbachol on c-fos mRNA and a cGMP analog mimicked carbachol. Carbachol 46-55 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 59-64 1415544-0 1992 CCK, bombesin, and carbachol stimulate c-fos, c-jun, and c-myc oncogene expression in rat pancreatic acini. Carbachol 19-28 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 39-44 1285949-4 1992 Although carbachol or trihexyphenidyl alone was ineffective in inducing c-fos mRNA, the combination of levodopa and carbachol (> or = to 0.1 mg/kg) significantly suppressed the induction of c-fos mRNA as compared with levodopa given alone. Carbachol 116-125 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 193-198 10187833-5 1999 In these studies we confirmed that H7, but not HA1004, potently blocks the induction of zif268 and c-fos mRNA by nerve growth factor, carbachol, phorbol ester, Ca2+ ionophore, or forskolin. Carbachol 134-143 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 99-104 9300429-6 1997 Carbachol activation of parafascicular neurons also induced the synthesis of c-Fos-like immunoreactive proteins in the pallidal neurons. Carbachol 0-9 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 77-82 8637618-0 1995 The association of thirst, sodium appetite and vasopressin release with c-fos expression in the forebrain of the rat after intracerebroventricular injection of angiotensin II, angiotensin-(1-7) or carbachol. Carbachol 197-206 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 72-77 8637618-1 1995 The effect intracerebroventricular injections of angiotensin II (0.1 nm), angiotensin-(1-7) (1 or 100 nm) and carbachol (500 ng) on c-fos expression was examined in the forebrain of Lister hooded rats. Carbachol 110-119 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 132-137 8584207-5 1995 Microinfusion of muscarinic agonist, carbachol, induced Fos expression as well, but mostly in the oxytocin neurons. Carbachol 37-46 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 56-59 8092325-0 1994 Comparison of fos-like immunoreactivity induced in rat brain by central injection of angiotensin II and carbachol. Carbachol 104-113 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 14-17 2572704-5 1989 Carbachol induced accumulation of mRNA for c-fos and the other TIS genes. Carbachol 0-9 Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus 43-48