PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28499878-5	2017	From in vitro experiments, we found that sulpiride is a substrate of hOCT1 (Km 2.6 muM), hOCT2 (Km 68 muM), hMATE1 (Km 40 muM), and hMATE2-K (Km 60 muM).	Sulpiride	41-50	latexin	Homo sapiens	83-86	
28499878-5	2017	From in vitro experiments, we found that sulpiride is a substrate of hOCT1 (Km 2.6 muM), hOCT2 (Km 68 muM), hMATE1 (Km 40 muM), and hMATE2-K (Km 60 muM).	Sulpiride	41-50	latexin	Homo sapiens	102-105	
28499878-5	2017	From in vitro experiments, we found that sulpiride is a substrate of hOCT1 (Km 2.6 muM), hOCT2 (Km 68 muM), hMATE1 (Km 40 muM), and hMATE2-K (Km 60 muM).	Sulpiride	41-50	latexin	Homo sapiens	102-105	
28499878-5	2017	From in vitro experiments, we found that sulpiride is a substrate of hOCT1 (Km 2.6 muM), hOCT2 (Km 68 muM), hMATE1 (Km 40 muM), and hMATE2-K (Km 60 muM).	Sulpiride	41-50	latexin	Homo sapiens	102-105	
28499878-6	2017	Moreover, the uptake of sulpiride by human hepatocytes was diminished by tetraethylammonium, and saturable with Km of 18 muM.	Sulpiride	24-33	latexin	Homo sapiens	121-124	
27260326-4	2016	EtOH (50 or 150 mg%) or the D2/3 receptor antagonist sulpiride (100 or 200 muM) was microinjected into the pVTA while DA was sampled with microdialysis in the NAC shell (NACsh).	Sulpiride	53-62	latexin	Homo sapiens	75-78	
27260326-7	2016	In contrast, sulpiride increased DA release in the NACsh more in the "Water" than "EtOH" groups (e.g., 200 muM sulpiride: to ~ 190-240 vs 150-160% of baseline).	Sulpiride	13-22	latexin	Homo sapiens	107-110	
27260326-7	2016	In contrast, sulpiride increased DA release in the NACsh more in the "Water" than "EtOH" groups (e.g., 200 muM sulpiride: to ~ 190-240 vs 150-160% of baseline).	Sulpiride	111-120	latexin	Homo sapiens	107-110	
22651101-4	2012	The MPTP-mediated transient AMPAr-fEPSP facilitation was antagonized by the dopamine D2-like receptor antagonists, eticlopride (1 muM) and sulpiride (1 and 40 muM).	Sulpiride	139-148	latexin	Homo sapiens	159-162