PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
22049425-8	2011	Importantly, all, IC-ERN, delta/theta power, and PES were modulated by COMT x Substance interactions such that the Val allele predicted elevated IC-ERN, delta/theta power, and PES after placebo; this association was reversed under sulpiride.	Sulpiride	231-240	catechol-O-methyltransferase	Homo sapiens	71-75	
24773408-1	2014	Consistent with dopamine accounts of internal and external feedback processing, prior work showed that the dopamine D2 receptor antagonist sulpiride modulates the relationship between the dopaminergic COMT Val158Met polymorphism and the error-related negativity (ERN).	Sulpiride	139-148	catechol-O-methyltransferase	Homo sapiens	201-205	
24773408-3	2014	N = 83 female participants genotyped for COMT Val158Met received 200 mg sulpiride versus placebo and performed a virtual ball-catching task.	Sulpiride	72-81	catechol-O-methyltransferase	Homo sapiens	41-45	
22049425-9	2011	Because low doses of sulpiride presumably increase PFC DA levels, the COMT x Substance interaction supports the hypothesis that low (Val, placebo) and high (Met, sulpiride) versus medium (Val, sulpiride; Met, placebo) DA levels elevate reactivity to errors.	Sulpiride	21-30	catechol-O-methyltransferase	Homo sapiens	70-74