PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19966860-7	2010	miR-29b locked nucleic acid (LNA) antisense oligonucleotide transfection into LMP1-expressing cells reduced miR-29b expression and consequently reconstituted TCL1, suggesting that LMP1 negatively regulates TCL1 through miR-29b upregulation.	Oligonucleotides	44-59	microRNA 29b-1	Homo sapiens	0-7	
30006350-9	2018	Targeted proteomic analysis of affinity-purified samples identified several proteins interacting with synthetic oligonucleotides mimicking miR-29b, but these proteins did not interact with miR-29a.	Oligonucleotides	112-128	microRNA 29b-1	Homo sapiens	139-146	
19966860-7	2010	miR-29b locked nucleic acid (LNA) antisense oligonucleotide transfection into LMP1-expressing cells reduced miR-29b expression and consequently reconstituted TCL1, suggesting that LMP1 negatively regulates TCL1 through miR-29b upregulation.	Oligonucleotides	44-59	microRNA 29b-1	Homo sapiens	108-115	
19966860-7	2010	miR-29b locked nucleic acid (LNA) antisense oligonucleotide transfection into LMP1-expressing cells reduced miR-29b expression and consequently reconstituted TCL1, suggesting that LMP1 negatively regulates TCL1 through miR-29b upregulation.	Oligonucleotides	44-59	microRNA 29b-1	Homo sapiens	108-115	
19850741-7	2009	Together, the data support a tumor suppressor role for miR-29 and provide a rationale for the use of synthetic miR-29b oligonucleotides as a novel strategy to improve treatment response in AML.	Oligonucleotides	119-135	microRNA 29b-1	Homo sapiens	111-118