PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 19966860-7 2010 miR-29b locked nucleic acid (LNA) antisense oligonucleotide transfection into LMP1-expressing cells reduced miR-29b expression and consequently reconstituted TCL1, suggesting that LMP1 negatively regulates TCL1 through miR-29b upregulation. Oligonucleotides 44-59 microRNA 29b-1 Homo sapiens 0-7 30006350-9 2018 Targeted proteomic analysis of affinity-purified samples identified several proteins interacting with synthetic oligonucleotides mimicking miR-29b, but these proteins did not interact with miR-29a. Oligonucleotides 112-128 microRNA 29b-1 Homo sapiens 139-146 19966860-7 2010 miR-29b locked nucleic acid (LNA) antisense oligonucleotide transfection into LMP1-expressing cells reduced miR-29b expression and consequently reconstituted TCL1, suggesting that LMP1 negatively regulates TCL1 through miR-29b upregulation. Oligonucleotides 44-59 microRNA 29b-1 Homo sapiens 108-115 19966860-7 2010 miR-29b locked nucleic acid (LNA) antisense oligonucleotide transfection into LMP1-expressing cells reduced miR-29b expression and consequently reconstituted TCL1, suggesting that LMP1 negatively regulates TCL1 through miR-29b upregulation. Oligonucleotides 44-59 microRNA 29b-1 Homo sapiens 108-115 19850741-7 2009 Together, the data support a tumor suppressor role for miR-29 and provide a rationale for the use of synthetic miR-29b oligonucleotides as a novel strategy to improve treatment response in AML. Oligonucleotides 119-135 microRNA 29b-1 Homo sapiens 111-118