PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
29367875-12	2018	Learning points: DM1 patients could be affected by a variety of multiple endocrinopathies.Our patients with DM1 presented rare combinations of multiple endocrinopathies; diabetes mellitus, combined form of primary and secondary hypogonadism and dysfunction of HPA axis.Testosterone treatment of hypogonadism in patients with DM1 could improve body composition.The patients with DM1 should be assessed endocrine functions and treated depending on the degree of each endocrine dysfunction.	Testosterone	269-281	immunoglobulin heavy diversity 1-7	Homo sapiens	17-20	
29367875-12	2018	Learning points: DM1 patients could be affected by a variety of multiple endocrinopathies.Our patients with DM1 presented rare combinations of multiple endocrinopathies; diabetes mellitus, combined form of primary and secondary hypogonadism and dysfunction of HPA axis.Testosterone treatment of hypogonadism in patients with DM1 could improve body composition.The patients with DM1 should be assessed endocrine functions and treated depending on the degree of each endocrine dysfunction.	Testosterone	269-281	immunoglobulin heavy diversity 1-7	Homo sapiens	108-111	
29367875-12	2018	Learning points: DM1 patients could be affected by a variety of multiple endocrinopathies.Our patients with DM1 presented rare combinations of multiple endocrinopathies; diabetes mellitus, combined form of primary and secondary hypogonadism and dysfunction of HPA axis.Testosterone treatment of hypogonadism in patients with DM1 could improve body composition.The patients with DM1 should be assessed endocrine functions and treated depending on the degree of each endocrine dysfunction.	Testosterone	269-281	immunoglobulin heavy diversity 1-7	Homo sapiens	108-111	
29367875-12	2018	Learning points: DM1 patients could be affected by a variety of multiple endocrinopathies.Our patients with DM1 presented rare combinations of multiple endocrinopathies; diabetes mellitus, combined form of primary and secondary hypogonadism and dysfunction of HPA axis.Testosterone treatment of hypogonadism in patients with DM1 could improve body composition.The patients with DM1 should be assessed endocrine functions and treated depending on the degree of each endocrine dysfunction.	Testosterone	269-281	immunoglobulin heavy diversity 1-7	Homo sapiens	108-111	
15855259-9	2005	Girls with DM1 at Tanner stage 5 had higher stimulated LH to FSH ratio, testosterone, and 17OHProg levels than girls at Tanner stage 4.	Testosterone	72-84	immunoglobulin heavy diversity 1-7	Homo sapiens	11-14	
18547959-6	2008	The adiponectin levels in the girls with DM1, but not in the controls, exhibited a negative correlation with OV (r = -0.427, p = 0.002) and testosterone (r = -0.262, p = 0.049).	Testosterone	140-152	immunoglobulin heavy diversity 1-7	Homo sapiens	41-44	
18547959-9	2008	CONCLUSIONS: Decreasing adiponectin levels observed during puberty in adolescents with DM1 correlate with increasing testosterone levels and OV.	Testosterone	117-129	immunoglobulin heavy diversity 1-7	Homo sapiens	87-90	
16569737-8	2006	DM1 women had higher levels of testosterone and androstenedione and larger ovarian volume and follicle number by ovary than C. PCOM was present in 54.8% of DM1 and 13.2% of C (P < 0.001).	Testosterone	31-43	immunoglobulin heavy diversity 1-7	Homo sapiens	0-3