PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 9813248-3 1998 We have shown that concurrent intraperitoneal administration of GM1 with a low and otherwise unprotective intracerebroventricular dose of nerve growth factor, can also prevent the loss of these fimbria fornix axotomised cholinergic neurons, where GM1 alone does not have this effect. G(M1) Ganglioside 64-67 nerve growth factor Homo sapiens 138-157 15312164-7 2004 GM1 elicited a time-dependent increase in nerve growth factor protein and mRNA in NIH-3T3 cells expressing TrkA or TrkC receptor but not in wild-type cells. G(M1) Ganglioside 0-3 nerve growth factor Homo sapiens 42-61 15312164-9 2004 The ability of GM1 to increase nerve growth factor mRNA levels was blocked by TrkC-IgG but not by TrkB-IgG receptor body. G(M1) Ganglioside 15-18 nerve growth factor Homo sapiens 31-50 9813248-4 1998 This study further confirms the neuroprotective actions of GM1 and suggests that it may interact to potentiate the effect of nerve growth factor on these axotomised septal cholinergic neurons. G(M1) Ganglioside 59-62 nerve growth factor Homo sapiens 125-144 9668353-4 1998 Presence of 20-microM exogenous GM1 for 4 days in NGF and PDMP containing cell cultures led to an increase of cell-associated GM1(15-fold), GM2 (10-fold), GM3 (15 fold), GD1a (4-fold), GD2, GD1b, and GT1b (all 3-fold), and partially reversed the PDMP (and FB1) effects on neurite growth and GAP-43 distribution. G(M1) Ganglioside 32-35 nerve growth factor Homo sapiens 50-53 9668353-4 1998 Presence of 20-microM exogenous GM1 for 4 days in NGF and PDMP containing cell cultures led to an increase of cell-associated GM1(15-fold), GM2 (10-fold), GM3 (15 fold), GD1a (4-fold), GD2, GD1b, and GT1b (all 3-fold), and partially reversed the PDMP (and FB1) effects on neurite growth and GAP-43 distribution. G(M1) Ganglioside 126-129 nerve growth factor Homo sapiens 50-53 9668382-0 1998 Ganglioside GM1 potentiates the stimulatory effect of nerve growth factor on peripheral nerve regeneration in vivo. G(M1) Ganglioside 0-15 nerve growth factor Homo sapiens 54-73 22484968-1 2012 In this mini-review I summarize our research efforts in ascertaining the possible neuro-reparative properties of the GM1 ganglioside and its cooperative effects with NGF in stroke-lesion models. G(M1) Ganglioside 117-132 nerve growth factor Homo sapiens 166-169 22496904-6 2012 The ganglioside, GM1, caused increased association of NGF, TrkA, and microtubules with DRMs, but a decrease in p75(NTR). G(M1) Ganglioside 17-20 nerve growth factor Homo sapiens 54-57 3919160-0 1985 Ganglioside GM1 does not initiate, but enhances neurite regeneration of nerve growth factor-dependent sensory neurones. G(M1) Ganglioside 0-15 nerve growth factor Homo sapiens 72-91 3919160-3 1985 However, the simultaneous presence of GM1 (100 micrograms/ml) and NGF (0.5-5 ng/ml) throughout a 5-day period of culture resulted in an enhancement of previously reported NGF-induced increases in the expression of neurofilament protein. G(M1) Ganglioside 38-41 nerve growth factor Homo sapiens 171-174 3919160-4 1985 Further, the addition of GM1 (0-200 micrograms/ml) at 48 h in vitro to cultures initially established in the presence of 5 ng/ml NGF substantially increased the subsequent expression of neurofilament protein, this response being both independent of and not potentiated by NGF. G(M1) Ganglioside 25-28 nerve growth factor Homo sapiens 129-132 3919160-4 1985 Further, the addition of GM1 (0-200 micrograms/ml) at 48 h in vitro to cultures initially established in the presence of 5 ng/ml NGF substantially increased the subsequent expression of neurofilament protein, this response being both independent of and not potentiated by NGF. G(M1) Ganglioside 25-28 nerve growth factor Homo sapiens 272-275