PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28654510-2	2017	The prime antiarrhythmic mechanisms of late INa inhibition have been suggested to be (1) suppression of intracellular calcium [Cai]-mediated rhythmic activity (through reduction in Cai secondary to the decrease in intracellular sodium [Nai]) and (2) normalization of repolarization.	Sodium Iodide	236-239	internexin neuronal intermediate filament protein alpha	Homo sapiens	44-47	
28654510-4	2017	Late INa influx may significantly contribute to Nai loading, but it seems to largely occur under the combined conditions of augmented late INa density, bradycardia, and prolonged repolarization.	Sodium Iodide	48-51	internexin neuronal intermediate filament protein alpha	Homo sapiens	5-8