PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32026338-1	2020	PURPOSE: In males, AMH is secreted by immature Sertoli cells; following exposure to endogenous androgens, Sertoli cells undergo a process of maturation which ultimately inhibits AMH expression to undetectable levels in the serum.	Androgens	95-104	anti-Mullerian hormone	Homo sapiens	178-181	
31735954-9	2019	PARTICIPANTS/MATERIALS, SETTING, METHODS: AMH protein production by cells exposed to androgens was measured by ELISA.	Androgens	85-94	anti-Mullerian hormone	Homo sapiens	42-45	
31735954-2	2019	SUMMARY ANSWER: Exposure of granulosa cells (GCs) to high levels of androgens, equivalent to that found in PCOS, prevented the fall in AMH and was associated with dysregulated AMH-SMAD signalling leading to stalled follicle progression in PCOS.	Androgens	68-77	anti-Mullerian hormone	Homo sapiens	135-138	
31735954-2	2019	SUMMARY ANSWER: Exposure of granulosa cells (GCs) to high levels of androgens, equivalent to that found in PCOS, prevented the fall in AMH and was associated with dysregulated AMH-SMAD signalling leading to stalled follicle progression in PCOS.	Androgens	68-77	anti-Mullerian hormone	Homo sapiens	176-179	
31735954-27	2019	The AMH decline may either be a direct effect of androgens or an indirect one via conversion to oestradiol and acting through the upregulation of ERalpha, which is known to stimulate the AMH promoter.	Androgens	49-58	anti-Mullerian hormone	Homo sapiens	4-7	
31725884-20	2019	Also, the relationships between intrafollicular AMH, gonadotropin and androgen levels in the same follicle support the hypothesis that FF AMH concentration may reflect granulosa cell proliferation during gonadotropin-stimulated follicle growth.	Androgens	70-78	anti-Mullerian hormone	Homo sapiens	138-141