PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 32054927-4 2020 Binding of Fis to the i-motif from the promoter region of the human vascular endothelial growth factor (VEGF) gene dramatically induced the excited state intramolecular proton transfer (ESIPT) reaction that significantly enhanced the intensity of the tautomer emission band of Fis. fisetin 11-14 vascular endothelial growth factor A Homo sapiens 68-102 32724364-0 2020 Fisetin inhibits vascular endothelial growth factor-induced angiogenesis in retinoblastoma cells. fisetin 0-7 vascular endothelial growth factor A Homo sapiens 17-51 32724364-13 2020 Thus, fisetin was found to inhibit angiogenesis via inhibition of the VEGF/VEGFR signaling pathway, and could be used as a candidate drug to inhibit angiogenesis in retinoblastoma. fisetin 6-13 vascular endothelial growth factor A Homo sapiens 70-74 32054927-4 2020 Binding of Fis to the i-motif from the promoter region of the human vascular endothelial growth factor (VEGF) gene dramatically induced the excited state intramolecular proton transfer (ESIPT) reaction that significantly enhanced the intensity of the tautomer emission band of Fis. fisetin 11-14 vascular endothelial growth factor A Homo sapiens 104-108 32054927-4 2020 Binding of Fis to the i-motif from the promoter region of the human vascular endothelial growth factor (VEGF) gene dramatically induced the excited state intramolecular proton transfer (ESIPT) reaction that significantly enhanced the intensity of the tautomer emission band of Fis. fisetin 277-280 vascular endothelial growth factor A Homo sapiens 68-102 32054927-4 2020 Binding of Fis to the i-motif from the promoter region of the human vascular endothelial growth factor (VEGF) gene dramatically induced the excited state intramolecular proton transfer (ESIPT) reaction that significantly enhanced the intensity of the tautomer emission band of Fis. fisetin 277-280 vascular endothelial growth factor A Homo sapiens 104-108 32054927-6 2020 As a result, the VEGF i-motif did not act as a replication block in the presence of Fis, which indicates the applicability of Fis for the regulation of gene expression of VEGF. fisetin 126-129 vascular endothelial growth factor A Homo sapiens 17-21 32054927-6 2020 As a result, the VEGF i-motif did not act as a replication block in the presence of Fis, which indicates the applicability of Fis for the regulation of gene expression of VEGF. fisetin 126-129 vascular endothelial growth factor A Homo sapiens 171-175 32054927-7 2020 The fluorescence and biological properties of Fis may be utilised for theranostics applications for cancers related to a specific cancer-related gene, such as VEGF. fisetin 46-49 vascular endothelial growth factor A Homo sapiens 159-163 22139440-3 2012 Fisetin (10-50 muM) strongly inhibited the regular serum plus growth supplement- and vascular endothelial growth factor (VEGF)-induced growth (up to 92%, P < 0.001) and survival (up to 16%, P < 0.001) of HUVEC in a dose- and time-dependent manner. fisetin 0-7 vascular endothelial growth factor A Homo sapiens 85-119 22139440-3 2012 Fisetin (10-50 muM) strongly inhibited the regular serum plus growth supplement- and vascular endothelial growth factor (VEGF)-induced growth (up to 92%, P < 0.001) and survival (up to 16%, P < 0.001) of HUVEC in a dose- and time-dependent manner. fisetin 0-7 vascular endothelial growth factor A Homo sapiens 121-125 22139440-6 2012 Furthermore, fisetin inhibited capillary-like tube formation on Matrigel (up to 85%, P < 0.001) as well as migration (up to 66%, P < 0.001), which were associated with decreased expression of endothelial nitric oxide synthase (eNOS) and VEGF in HUVEC. fisetin 13-20 vascular endothelial growth factor A Homo sapiens 243-247 20153296-4 2010 We found that apigenin, luteolin, fisetin and quercetin inhibited hypoxia-induced VEGF expression in the low micromolar range. fisetin 34-41 vascular endothelial growth factor A Homo sapiens 82-86 20153296-6 2010 Interestingly, only a group of VEGF inhibitors, including apigenin, flavone and 4",7-dihydroxiflavone, reduced the expression of HIF-1alpha under these conditions, whereas others, such as fisetin, luteolin, galangin or quercetin, induced HIF-1alpha expression while reducing those of VEGF. fisetin 188-195 vascular endothelial growth factor A Homo sapiens 31-35